The main directions of preventing tooth decay

June 9, 2024
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The main directions of preventing tooth decay. General and local caries factors. Endogenous and exogenous prevention of dental caries. Means and methods efficiency evaluation.

 

Dental caries, also known as tooth decay or a cavity, is an infection, bacterial in origin, that causes demineralization and destruction of the hard tissues (enamel, dentin and cementum), usually by production of acid by bacterial fermentation of the food debris accumulated on the tooth surface.[1]If demineralization exceeds saliva and other remineralization factors such as from calcium and fluoridated toothpastes, these hard tissues progressively break down, producing dental caries (cavities, holes in the teeth). The bacteria most responsible for dental cavities are the mutans streptococci, most prominently Streptococcus mutans and Streptococcus sobrinus, and lactobacilli. If left untreated, the disease can lead to pain,tooth loss and infection. Today, caries remain one of the most common diseases throughout the world. 

O.V.Udovitska (1987) selects 7 basic signs the risk of caries: 1) viscid saliva; 2) acid reaction of environment of cavity of mouth; 3) propensity is to the superficial deposit of dental deposit at the normal hygienical mode;4) hypoplasia enamels of teeth as index of vicious development of enamel; 5) premature eruption of teeth (on a half-year and more — temporal, on a year and more — permanent); 6) and the inherited inclination; 7)toxicosis pregnancies of mother.

Operate these factors it is easier to find out, if to examine the normal state of enamel as a dynamic equilibrium between the permanent processes of de- and remineralisation. In that case, when in dental fabrics the processes of demineralization prevail above remineralisation, there is an area of demineralization as a carious spot. Subsequent progress of process of demineralization enamel and dentine results in formation of carious cavity.

 

 

Resistance of dental fabrics is broken

The structure of enamel acts important part in the origin of caries. Homogeneity of its structure rises as far as ripening of enamel, there is smoothing of relief of surface, without prisms areas which mask the heads of prisms appear. Narrowing of scopes of prisms, decline of contrasting of Rettsius’s lines is marked in the proper layers, the volume of mісro spaces diminishes to 0,1—0,2 %, that brings enamels over to multiplying a closeness. The amount of water diminishes in an enamel. Due to entering of ion of fluorine enamel maintenance of fluorine apatites is multiplied in her that promotes her caries resistance. The aggregate of age-old processes which take place in an enamel reduces its mісro porosity and accordingly, and permeability, and promotes caries resistance.

In ripening of enamel an important role belongs to the fluorine the amount of which after eruption of tooth is gradually multiplied. His including is proved from saliva in an enamelA fluorine regulates the process of absorption of calcium hard fabrics of tooth. Speed of mіneralіzation considerably grows in presence a fluorine. Even during such low concentration of fluorine as a 1:1000 speed of mіneralіzation grows in 3—5 times.

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For determination of resistance of enamel of teeth to the caries apply the test of enamel resistence (TER-TEST); V. R. Okushko, L. I. Kosareva, 1983) He allows to set functional resistance of enamel in relation to acid. A test can be used as initially-diagnostic and also for the objective estimation of efficiency of remineralization therapy during a clinical supervision and treatment of patients. The method of conducting of test is such:

conduct the professional hygiene of mouth cavity

the crowns of the teeth process the distilled water and wadding tampon, dry out

on the vestibular surface of central overhead chisel inflict the drop of a 1% solution of HCl diameter no more than 2mm.

through 5sec acid is washed off the distilled water

the crown of the tooth is dried out a wadding tampon

on a surface crowns inflict a tampon, moistened in 1% water solution of metilen dark blue

take off dye one motion of wadding tampon from the surface of enamel

estimate a test after the special to 10 balls color scale

on an area where mіneralіzation inflict varnish with fluorine

In obedience to 10 balls scale of dark blue color determine the degree of resistance of teeth to the caries:

1-3 marks an area is painted in a pale blue color which determines considerable structurally-functional resistance of enamel and high firmness of teeth to the caries

4-6 marks an area is painted in a blue color which determines middle structurally-functional resistance of enamel and middle firmness of teeth to the caries

7-9 marks an area is resistance in a dark blue color which determines the decline of structurally-functional resistance of enamel and high degree of risk of origin of caries

10 marks an area is painted in a navy which characterizes structurally-functional resistance of enamel and maximal risk of origin of caries is extremely reduced.

An inferior in a high-quality and quantitative relation feed results    in    the row    of violations    in    the systems    of carbohydrate    and mineral exchange which sharply loosens resistance of hard fabrics of teeth to the carious factors.

It is bound   a promoted   cariogenic meal   to   surplus maintenance in the rations of carbohydrates, diminishing mineral salts by vitamins and other vitally by important for an organism components. Thus consider the promoted use of refined sugars the basic starting mechanism of development of decay of teeth, development of specific microflora and decline of acidity of cavity of mouth, which is a condition which promotesdemineralization of enamels.

Except for composition of rations, in development of caries a large value is given the so-called civilization of feed, which is accompanied the high degree of culinary treatment of meal which diminishes the functional loading on a masticatory vehicle.

It is thus shown many authors that defeat caries mainly: it is related to maintenance of fluorine in water and less measure — in food stuffs, as at the last on territory different districts the level of fluorine hesitates insignificantly. Found out especially high staggered a caries for persons which use water with the very low concentrations of fluorine (at the level of 0,1…0,3 mg/l)

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A considerable place in the origin of caries belongs the carried and concomitant disease. It is set that for adults and children which carried sharp infectious diseases or are ill chronic illnesses of internal organs and systems, especially often there is a decay of teeth.

From the extreme influencing which result in the intensive defeat of hard fabrics of tooth, it follows to specify a radiation on ionizing. As numerous clinical supervisions testify x – ray therapy which is often enough used in a clinic is always accompanied the defeat of teeth regardless of area which tested an irradiation.

Role of saliva.

Most reliable is possibility of change of properties and will make enamels through saliva. Character of saliva selection, quantitative and high-quality indexes, change at some diseases of commons.

All physiology processes of hard fabrics of tooth after eruption flow in interrelation with saliva — biological environment of cavity of mouth.

Basic aspects of participation of saliva in support of homergy of mineral matters in the enamel of teeth followings:

1. The mineralization function of saliva, due to which mіneralіzation of teeth, “ripening” of enamel is carried out after eruption, is supported optimum composition of enamel, there is his renewal.

2. Protective function which consists in protecting of cavity of mouth from the pathogenic action of factors of external environment.

3. Clearing the role of saliva which consists in the permanent mechanical and chemical cleaning of cavity of mouth from tailings of meal, microflora, etc.

Consequently, in physiology terms saliva is the supersaturated solution on maintenance to the calcium and phosphate.

The state of supersaturating of saliva of hydroxide apatites has an important value for saving and support of constancy of dental fabrics in the cavity of mouth, for providing of homergy of mineral components. Supersaturating of saliva, from one side, hinders salts of calcium and phosphate dissolution of enamel, as saliva is already supersaturated such which make an enamel components; from other side, instrumental in diffusion in the enamel of ions of calcium and phosphate, as their active concentration in saliva considerably exceeds such in an enamel, and the state of supersaturating is instrumental in their adsorption on an enamel.

The maiatural regulator of homergy in the cavity of mouth is pH of saliva. In the norm of pH saliva hesitates within the limits of 6,8 — 7,2. The considerable decline of pH saliva, which is contained during 30 mines andmore, is marked after adopting carbohydrates. The decline of pH saliva creates direct influence on its mineralization function she becomes calcium – by a deficit that from mineralization becomes demineralization liquid.

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Dental deposits

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On permeability of enamel superficial educations have substantial influence on teeth. They are divided on:

–    no mineralized: pellicle, dental deposit, dental name – plate  

–    mineralized: above- and under gingival dental tartar.

 

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Pellicle

Pellicle— organic tape which appears after eruption of tooth. She has the appearance of dark grey tape, most on the area of fissure, the least – on humps. Pellicle is deprived bacteria. Something later gramme positive cocci penetrate to her. Then colonization goes gramme negative cocci and anaerobes. On their cellular surface there are receptors which have a cognation to glycoproteins. Pellicle protects safety of enamel due to deceleration of transport of ions of calcium and phosphorus to the enamel. The changes of properties of pellicle (structures, composition) can brake or stimulate development of caries. She can be taken off from a surface of enamel only by facilities of abrasives. After its removal at presence of contact of tooth with saliva of pellicle recommences during 20—30 min.

Pellicle acts considerable part in the processes of diffusion in the superficial layer of enamel, giving the last selective permeability. In additionpellicle protects an enamel from the action of matters, that her dissolve.

The thickness of pellicle on the separate areas of crown   hesitates from 1—2 to 10 mcm. The thinnest she wherein teeth clear up well, and most shown in fissures, oear neck and lateral surfaces of crown.

Pellicle of tooth is instrumental in attaching to his surface of microorganisms and formation of dental name-plate.

Dental name-plate

A dense dental raid or dental name-plate for the exposure of which coloring is needed the special solutions takes place above pellicle. A name-plate takes place above and under gums, more frequent in near neck area andfissures. Formation of name-plate begins from tacking of bacteria to pellicle or to the surface of enamel. Microorganisms register to the tooth by sticky interbacterial matrix. The amount of raid is certain appears already in 2 hours after the careful cleaning of teeth. The maximal accumulation of name-plate takes place during 30 days.

A dental name-plate consists of microorganisms, cages of epitheliums, leucocytes and macrophage. Bacteria are 10% hard remain of name-plate, all other is intercellular matrix.
 Intercellular matrix of raid contains organic (squirrel, polysugars, fats) and inorganic (calcium, phosphorus, potassium, sodium, fluorine)   matters. Carbohydrates present in matrix are presented by dextran   — polysugar  what is formed by bacteria.   He   appears by streptococci from sugar.

A name-plate grows due to permanent stratification of new bacteria. As far as growth of name-plate its mіkrobial flora changes from predominance of cocci (mainly gramme positive) to more difficult population with maintenance of stick similar microorganisms.

At first a name-plate consists of characteristic cocci. Streptococcus are 50% bacterial flora with predominance of Str. mutans which has most carious action. To the extent of that how a name-plate is thickened, inwardly her anaerobic terms are created and a flora changes accordingly. The source of feed of superficial microorganisms, for certain, a mouth environment is. Deep bacteria for the growth use the metabolic products of other bacteria of name-plate and components of its matrix.  It leads to that on a 2—3th day gramme negative   cocci   and   sticks appear in a name-plate,    on   4—5th   —   fusobacteria,    the amount of anaerobes is sharplymultiplied.  From 30 to the 90th day the amount of streptococci diminishes to 30—40% and the number of stick similar are multiplied to 40%.

Consequently, during 1—2 days of formation of name-plate in her prevails Str. mutans, his carious action determined making them of plenty of suckling acid in presence carbohydrates. It results in the local falling of pH (to 5,0) directly on the surface of enamel under a dental name-plate and to the increase of permeability of enamel. At the protracted support of critical level of ions of hydrogen there is acid dissolution in the least proof areas of enamel (interprismatic matter, Rettsius lines) with subsequent penetration of acids in the subsuperficial layer of enamel and her demineralization. Subsequent formation of organic acids on the surface of enamel strengthens the processes of demineralization that results in formation of micropore in an enamel, filled microorganisms and products of their metabolism. The amount of anaerobes grows thus.

Certainly that than higher speed of formation of dental name-plate the more so expressed her cariogenic action. For children which have the promoted index of dental name-plate, the increase of intensity of caries takes place in three times quick than for children with a low index. Except for it caries for children develops in those cases, if in croflorof dental name-plate prevails Str. mutans.

However much numerous factors influence on cariogenic action of dental name-plate, in particular, croflora of cavity of mouth, amount of carbohydrates which are used, quantitative and high-quality composition of saliva, its physical and chemical properties, maintenance in her mineral components: than higher level of calcium and phosphorus, the less cariogenic potential of name-plate.

A dental name-plate on the surface of tooth mechanically insulates an enamel from the action of saliva.

Sugars is instrumental in formation of name-plate, reproduction of microorganisms which then use them for about presentation it cariogenic actions.

A soft dental deposit is yellow or grey – white soft and sticky deposit which not densely adjoins to the surface of tooth. He is visible without coloring the special solutions and can be partly washed off the stream of water. Accumulates in a night-time especially for persons which do not carry out the regular care of cavity of mouth.

This conglomerate, unlike a name-plate, does not have a clear structure. Microbes enter in his composition, the cast – off epithelium of mucus shell, leucocytes, and also fats and proteins of salivaA soft deposit settles not only on the surface of teeth but also on the mucus shell of gums, especially in the places of their bad self cleaning. This type of deposits assists development of inflammatory processes of margіnal paradontium under act of local irritating factors — toxic products of vital functions   of microorganisms   of soft  dental   deposit. Tailings of meal stay too long in the cavity of mouth, especially in intradental intervals. Sticky products are wares of flours and pastry cooks, porridges — can stick and on other surfaces of teeth and to remain there on a few hours. In the conditions of cavity of mouth there is fermentation, rotting or disintegration of tailings of meal and products which are selected as a result of these processes stimulate the vital functions of microorganisms of cavity of mouth, growth of dental name-plates, influence on displacement of pH of mouth liquid.

 

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Soft dental deposit

 

Dental tartar it a dental name-plate mineralized in essence.

There is super gingival and sub gingival. On 90 % consists of inorganic remain among which basic seat is taken by salts of calcium.

Dental stone, — it dental name-plates are in essence mineralized. Their mіneralіzatіon takes place as a result of increase of pH of mouth liquid, and, fall in sediment of salts of phosphorus, calcium. Stagnation of saliva can promote to it. Before the change of pH name-plate the vital functions of microorganisms are drawn also. By a mineral spring there is mouth liquid super gingival tartar, and sub gingival — gingivalMіneralіzatіon of name-plate consists in binding by the protein-polysugars complexes of name-plate of salts of calcium. The concentration of calcium in a mineralized name-plate can in 10 times exceed its maintenance in saliva. Education a tartar depends on character of meal: a soft meal accelerates this process and the hard brakes him.

Mіneralіzation of name-plate begins on its surface which she is fastened to the tooth from matrix, and then takes microorganisms. As a rule, a dental stone is covered no mineralized name-plates. Education a stone lasts to the size, to characteristic for every individual.

 

 

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Super gingival tartar takes place above the gingival edge of mucus shell, densely adjoining to the surface of tooth. He shows by itself hard or clay similar formation of rather yellow-white color. The color of stone can change under act of dyes which are in food products. More frequent in all he is localized on the tongue surface of lower frontal teeth and on the surfaces of cheeks of lateral teeth which are placed opposite the conclusion channel of parotid of salivary gland.

Sub gingival it follows to examine a dental stone as a result of inflammatory process in regional paradontіum, caused more frequent in all a name-plate, because in connection with inflammation in tooth-gingival sulcus the amount of gingival liquid grows with the greater concentration of mineral salts. Sub gingival tartar is localized in gingival sulcus that under the edge of mucus ash and adjoins to the surface of tooth more densely than super gingival. Consistency of him is also denser, color — from rifle-green to the umber. Finding out sub gingival tartar is possible only by sounding of tooth-gingival connection. This type of mineralized dental deposits meets for children considerably rarer than super gingival. The last appears for children (35—70%), as a rule, not early than 9—10-years-old age.

A dental tartar contains to 90% inorganic components as crystals of salts of calcium (phosphate, carbonate), magnesium and row of other microelements. Organic part of the tartar are carbohydrates, proteins, fats of microorganisms, leucocytes, cast – off epithelium. In a sub gingival tartar found out salivary squirrel, and correlation of calcium and phosphorus considerably higher than in super gingival.

 

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TOOTH-PASTES

Now different tooth-pastes are the most widespread mean for the care of cavity of mouth.

They are folded usually

–    abrasive filler

–    linking component

–    superficially-active matters

–    antiseptic – preservative.

 

 

 

Component parts

Components                            

 

      

         Abrasive filler

Chalk (carbonate of calcium), aeroseel, silikgelalumogel,alumosilikat, 2 calcium – phosphate, 3 calcium – phosphate,pirophosphate calcium, insoluble metaphosphate sodium,kaolin, synthetic resins, polymeric connections ofmetilmetacrylat

     Moistening

Glycerin, polyethilenglikolsorbitol

  Gel formative linking

              matter

Sodium of karboksilmethiltselulozaoksiethiltselyuloza,koragenatalginat of sodium

    

Antiseptic, preservative

Benzoat of sodium, tetraborat sodium, boric acid, nipagin,nipazolparaformal’degid, ethers of laraoksibenzoil acid, chlorgeksidintriklozanpropilparaben

         Suds formative                                                                                             matters

Laurilsulfat of sodium

Matters which improve       qualities of tastes

Xilіt, xilitansorbіt, saccharin

     Aromatizator

Essential oils to crumple and other sharply smelling plants; synthetic  aromatizators

         Dye

Tartrazinrodamin, cosmetic dark blue

           Water

 

Biologically active matters which influence on mіneralіzatіon of fabrics of tooth

Fluorides, separate microelements and polymineral complexes, extracts of medical plants, enzymes, separate vitamins and their complexes

 

Abrasives fillers

Setting of matters of abrasives consists in cleaning, polishing of surface of tooth and in providing of viscidity to pasture. Pastes have cleansing ability higher with a high abrasive, polishing — with low. It was also set that the matters of abrasives can react with inorganic connections of enamel of tooth.

In more early standards of tooth-pastes connections of calcium were used in this quality. By a classic abrasive a chalk was chemically besieged.

A chalk (carbonate of calcium) is presently used rarely, as this abrasive is badly combined with medical additions and owns a large wearing away action. Modern tooth-pastes on chalky basis (Perlі, Phosphodent,Ftorodent, Extra) contain a high-quality chalk with low maintenance of oxides of aluminium and iron.

Silicic connections as systems of abrasives of tooth-pastes began to be used from the end of 70th, that at the beginning of development of silicic technology. Dioxide of silicon is well consonant with connections of fluorine and other active components, owns the controlled abrasive, that allows create to pasture with the wide range of the set properties. He also provides optimum pH — 7, due to which to pasture on the basis of dioxide of silicoormalize acid – alkaline balance. Dioxide of silicon (SiO2xH2O) is more frequent used in an amount 15—25 % on weight.

Presently there is a wide enough assortment of the so-called without abrasives facilities — gel similar of transparent tooth-pastes, got on the basis of connections of oxide of silicon, treated the special method. Gel similarto pasture own high suds formative ability, have a pleasant taste and attractive original appearance. In addition, due to formation of underlying structures of waters, gel allows to plug in his composition chemically incompatible matters, as a water shell hinders a chemical reaction between them. At immaturity or promoted abrasion of hard fabrics of teeth the low abrasive of gel similar pastes does them the best.

Except for these matters paste can contain also and medical  prophylactic additions (provitamins and vitamins, extracts and extracts of different medical plants, salt, microelements, enzymes and other matters).

 

Linking gel similar agents

Used for the receipt of homogeneous past similar consistency of tooth-paste. They can be natural and synthetic.

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Moistening

Provide a moisture retaining effect. Polyatomic alcohols are glycerin (15—25 % on weight), polyethilenglikol — used in composition tooth-pastes for the receipt of plastic mass: these connections are instrumental in saving, retaining of moisture in paste at its storage, promote the temperature of its freezing, multiply stability of suds which appears at cleaning of teeth, improve qualities of tastes to pasture. For retaining of moisturesorbitol is often used.

Other constituents of tooth-paste

As taste addition is often used 0,1 % to the saccharin. The sweetness of saccharin in 400—500 times exceeds the sweetness of sugar. Sodium salt of saccharin is used quite often.

Sorbіt turns out at proceeding in fructose. His sweetness in 2 times less, than sweetness of sugar.

Xilіt is a product of metabolism in an organism. Contained in plants, fruit (strawberries), vegetables (to the bow, carrot). Does not assimilate most types of microorganisms. Products with xilіt are not added microbiological decomposition. The sweetness of xilіt in 2 times exceeds the sweetness of sugar. Owns pleasant taste and creates the cool feeling in a company. Stimulates making of saliva, what instrumental inremineralization of enamel.

The role of aromas is carried out by mint butter, eucalyptus, mentollemon butter (not more than 1,5 %).

Active agents of tooth-pastes

More frequent in all the matters of the followings groups enter in the complement of tooth-pastes:

– fluorides

– connections of calcium

– phosphates

– complexes cro- and makroelements

– antiinflammation agents

– styptic agents

– biologically active matters

– enzymes

– antimicrobial agents

– mineral salts

– inhibitors of crystallization

– connections which reduce the sensitiveness of hard fabrics of teeth.

Fluorides

A fluorine is one of basic operating matters,  the mechanisms of influencing of which are related to diminishing of solubility of minerals of enamel, inhibition of formation of acids the bacteria of dental raid and participating in the processes of remineralization. Even the small concentrations of fluorine reduce the degree of demineralization enamel considerably, and that is why the frequent repeated use of preparations with the low concentration of fluorine is one of the most effective facilities of prophylaxis of caries.
A table of contents of fluorine in tooth-pastes, intended for the use children, must be not less 0,1% ions of fluorine that a considerable anticarious effect was  attained. On the bristle of tooth brush part is squeezed out from a tube to pasture, on volume proper the size of pea. Paste is spread for the surfaces of teeth before the beginning of their cleaning. During cleaning of teeth paste aim not spitting and after cleaning during one minute teeth are rinsed paste which remained in a company, and two-bit of water.
After cleaning of teeth it is not recommended to rinse the cavity of mouth and also it follows during one hour to avoid adopting a meal and drink.
Thus concentration of fluorine in saliva and in interdental intervals remains on possibility high that reduces the increase of 
proksimal caries in same queue. It follows to consider multiplying an amount the lack of this technique to pasture, that swallow.




 

  Triklozan

Triklozan creates the antimicrobial influencing in a wide spectrum. He is already a long ago used in different cleansers as addition. The most frequent concentration of triklozan in tooth-pastes is 0%. Triklozan multiplies permeability of shells of bacterial cages, that in same queue results in the exit of cytoplasm from a cage and its death. In the low concentrations of triklozan do inhibition synthesis of protein and metabolism of glucose ofbacteria.
Triklozan owns a antiinflammation action also. In researches, conducted on a present tense, it was not marked considerable effects of sides of triklozan.

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All tooth-pastes must satisfy such requirements.

– to be characteristics  (to clear ability the surface of teeth from a soft dental deposit and food tailings) of abrasives, it can be attained due to mechanical, washings or combination those and other properties.

– it is not enough to influence on physiology floraof cavity of mouth.

– during two years after an issue must not contain pathogenic   microorganisms   and   be   a nourishing   environment   for the vital functions of bacteria which will be able to penetrate to pastes in the process of the use.

– not to have sharp and chronic toxicness, irritating actions.
Now perfume – cosmetic industry makes the considerable enough assortment of tooth-pastes which it follows to divide into two large groups. Basic part of industrial production is made by hygienical tooth-pastes, intended for cleaning of teeth and cavity of mouth from tailings of meal and soft deposit. The second group is made by medical 
 prophylactic tooth-pastes to which as additional components enter useful biologically-active preparations. To pasture these except for a cleansing and refreshing action have favourable enough influence on fabrics of teeth, mucus shell of cavity of mouth, complementing medical measures.

Classification of tooth-pastes.

I. On the method of action

1. Hygienical — intended exceptionally for the delete of dental deposits.

 

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2. Medical  prophylactic — that take off those or other factors which are instrumental in the origin of diseases of teeth and fabrics of paradontium.

3. Medical are including active components which influence directly on a certain pathological process in the cavity of mouth.

 

II. For belongings

1. Child (from 1 year to 6)

2. Child – juvenile from 6

3. Juvenile (from 9)

4. Mixed

5. Adult

IIIOn the degree of suds education

1. Not foamy

2. Poorly foamy

3. Moderately foamy

4. Strongly foamy

IV. On maintenance of elements

1. Without active elements

2. With active elements:

a) fluorine containing

b) calcium containing

c) mixed

d) salts

e) anthiseptic

f) extracts of herbages

V. On maintenance of connections of fluorine

1. Fluorine free

2. Fluorine containing

a) one connection of fluorine

b) different connections of fluorine (is mixed fluorine and calcium containing)

 

VI On concentrations

1. With the very low concentration of fluorine

2. With the low concentration of fluorine

3. With a moderate concentration

4. With high

VII. On the actions of abrasives tooth-pastes subdivide into 3 basic a kind:

1. Anti-Plaque: middle degree of abrasive which hinders education
dental deposit.

 

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2. Type    of “Sensitive”: owns very much the low degree of abrasive for an account low   dispersion   of particles   (children   and   juvenile).   Used   people with the promoted sensitiveness of teeth.

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3. Type of “Smokers”: high and ever-higher degree of abrasive. Them it is impossible to apply often, no more 1st time per a week. Mainly they are used for professional cleaning of teeth.

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In same queue modern medical prophylactic tooth-pastes on the orientation of action and on composition it is possible to subdivide into the followings groups:

1. That influence on mіneralіzation of fabrics of tooth contain:

a) connection of fluorine

b) connection of calcium

c) phosphates (in particular hydroxide apatites)

d) complexes macro- and microelements (remodentegg-shell, complexes of salts).

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2. That influence on fabrics of paradontium and mucus shell of cavity of mouth contain:

a) antiinflammation agents

b) styptic agents,

c) biologically active matters (vitamins, extracts of medical plants)

d) enzymes

e) antiseptics

f) mineral salts.

 3. That reduce formation of dental name-plate contain:

a) antiseptics

b) mineral salts

c) enzymes

d) connection of fluorine.

4. That reduce education a tooth a stone contain:

a) inhibitors of crystallization

b) matters of abrasives.

5. That reduce the sensitiveness of hard fabrics of tooth contain:

a) connections to potassium

b) connections of strontium

c) formalin

 

6. Those, that bleaching contain:

a) inhibitors of crystallization

b) matters of abrasives

c) peroxides connections.

 

All bleaching tooth-pastes can be divided into two groups:

– to pasture, that chemical matters contain bleaching, for example, of peroxides connection, acids or enzymes.

– to pasture, that provide the high-quality delete of superficial dyes and polishing an effect due to the increase of abrasive. To pasture these does not operate at a good hygiene. That, if there is not a raid on teeth, and a dark color is a color of enamel, abrasive paste will not help. To pasture such good for smokers, for example.

Who suffers the disease of paradontium, pasturing such is better not to apply. They also contra-indicated persons with a hypersensitiveness and pathological abrasion.

 

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On the method of application medical  prophylactic tooth-pastes can be subdivided on:

1. To pasture for the daily cleaning of teeth.

2. To pasture and gels for valid for one occasion application in the certain intervals of time.

3. Gels are for appliques or easy embrocation after cleaning of teeth.

 

Children’s tooth-pastes

From adults they differ the less concentration of active components, low abrasive and also taste. Unlike adults which give advantage mint taste, for children he causes vomit reflex often. That is why in tooth-pastes of children’s more frequent use neutral or fruit aromatizators. Tooth-pastes of children’s are subdivided into two groups: for children to 6, which the concentration of fluorides is reduced in; and for children from 6 to 12-14, in which the concentration of fluorid is close to adult tooth-paste and an abrasive is saved reduced.

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Tooth-pastes with soda

It is the special group of pastes they contain a food soda (from 7 to 10%). Its properties: promotes clearing property to pasture, being not an abrasive; creates a weak alkaline environment in the cavity of mouth approximately on 20 minutes after cleaning of teeth; antiseptic action; creates a hypertensive environment, as a result takes off an edema from gums (well during pregnancy, in complex therapy of paradontitis); causes death of microbes, accountable for the origin of caries.

For associative perception between taste and color, there are the followings conformities to the law:

a) green color – moderately mentol;

b) the brilliant is saturated dark blue – as  a mint;

c) navy blue mat – as salt;

d) green dark – as vegetable;

e) orange-gold – as calendulas;                    

f) rose – as strawberry;

g) red – raspberry – as a raspberry and etc

Optimum, if you will choose paste on a reception for dentistry. It is possible to lick this problem, remembering that cheap pastes from unknown producers not so good, as firms. Tooth-pastes it follows to be on duty. Yes, at propensity to the caries and paradontitis it follows to apply anticarious paste in the morning, and in the evening – antiinflammation. It is important here to observe the correct technique of cleaning of teeth.

 All measures on the prophylaxis of caries of teeth it is possible to divide into state, social, medical, hygienical and educating.  The system of state preventives includes measures on the health protection mother and child, guard of environment. Realization of the state system of measures of health protection     population     is directed     foremost     on     the antenatal prophylaxis of diseases, on forming and   development of healthy child, on support of health of the grown man is basis of prophylactic direction of health protection in our country.

The system of social measures on the prophylaxis of caries is related to providing of healthy way of life is observance of the rational mode of labor, rest, scientifically grounded norms of feed, personal hygiene.

To the hygienical measures hygienical education of population from the questions of dentistry, control after the state of environment and feed, belong on the prophylaxis of caries. By introduction of hygienical measures (fluorination of drinking-water, control after high-quality and quantitative composition of meal and water) the necessity of children and adults is provided in the valuable factors of health of organism and organs of cavity of mouth.

Educating measures on the prophylaxis of caries of teeth are taken to diffusing learning about the healthy way of life, inoculating of skills of care of organs of cavity of mouth and support them in the healthy state.  These tasks are decided by doctors, middle   medical   personnel teachers and parents.

Medical measures on the prophylaxis of caries of teeth are directed for development and introduction etiologic and the nosotropic grounded facilities and methods of influence on an organism and organs of cavity of mouth for the increase of their firmness to the caries, and also on the decline of cariogenicity of unfavorable factors of environment on the cavity of mouth.

It follows to begin the prophylaxis of teeth decay in a period forming of organic matrix of the temporal and second teeth, their mіneralіzation and continue after teething to the complete ripening of enamel.

 

PROPHYLAXIS OF CARIES OF TEETH

Without medical prophylaxis of caries of teeth

Rational feed.

The process of forming and mіneralіzation of teeth begins in the embryo period of life of child and proceeds after its birth that is why for forming of teeth resistance to the caries there is important the high-quality feed of expectant mother and child.

The necessity of pregnant makes: to the 1,5 g calcium, 2,5 g phosphorus, 3 mg fluorine, 2,5 mg vitamin of B 5000—10000 МО of vitamin Don days. The necessity of consumption of these matters especially grows during the second half of pregnancy. In this period and in a period feeding of child a woman must use milk products — curd (no less as 200 g on a day), kefir, sour milk thick. Oligoelementss are contained in a beet, cabbage, nuts, oarweed, meat of rabbit, saltwater fish.

During all pregnancy the organism of woman needs the doubled amount of vitamins. The vitamin of C is especially important, vitamins of group B, A, D, E. Vitamin D takes part in formation of bone skeleton, regulating acalcium and phosphoric exchange, activity of ductless glands. At insufficiency of him a mineral exchange is violated in an organism, the amount of calcium and phosphorus diminishes in bone fabric, teeth cut throughlately, put must propensity to the caries. Day’s necessity of pregnant in the vitamin D is 500,0 МОVitamin A assists to correct development of bone fabric, provides normal activity of organ of sight, promotes firmness in relation to the diseases of mucus shells. Day’s dose of vitamin A — 2 mg.

For the children of 1st life milk of mother is an ideal product. It contains in optimum amounts and correlations biologically valuable albumens, fats, carbonhydratess, vitamins, mineral matters,    hormones,    immune   bodies,    enzymes    antimicrobial    and

bifidogenous factors.

A necessity in salts of calcium grows as far as growth and development of organism of child, vitamins, albumens, fluorine, which can be satisfied by the increase in the ration of milk products, green-stuffs, fruit.

 Strengthening of somatic health.

It is assumed that under act of commons diseases the terms of forming and ripening of hard fabrics of tooth change in the first turn of enamel which does them less proof in relation to influencing of cariogenic factors.

At the children of different age, burdened by the carried or concomitant diseases of internalss caries of teeth develops especially often. Thus a process is characterized by sharp motion. Practically the defeats of any organs and systems of child’s organism are extrapolated on hard fabrics of teeth. That is why it is needed with the purpose of prophylaxis of dental diseases, on possibility, to treat be – what somatic pathology.

Intensive mastication.

The large value in a prophylaxis has active   mastication,   valuable   loading on   maxillufacial area.   It is expedient  to promote  physiology  activity  of organs  and fabrics of cavity of mouth, especially at children, by multiplying a number and varieties of natural irritants, which at the permanent use of the treated meal becomes all less. The amount of children which are characterized by untrained of masseters through protracted their underloading under act of the developed “lazinesses of mastication”As a result putting such is renounced hard meal, is chewed languidly and slowly. Such children cost to recommend the use of products without previous culinary treatment (raw green-stuffs, fruit).

Medical prophylaxis

A medicinal prophylaxis is divided into general (endogenous) and local (exogenous). The endogenous in the turn is specific and heterospecific. At a specific prophylaxis preparations of fluorine are used and at heterospecific are vitamins, calcium containing preparations, roborants. The local consists in introduction to the cavity of mouth or directly on the surface of tooth of preparations of fluorine or remineralization solutions.  

Preparations of fluorine now are basic facilities of prophylaxis of teeth decay. The mechanism of protective action of fluorine on enamel consists in the assistance to the delay of phosphoric-calcium connections in an organism and processes of remіneralіzation of hard fabrics of tooth and also braking of activity of bacterial enzymes in the cavity of mouth and dental deposit.

Modern information show that the favourable action of fluorine is predefined by a few mechanisms:

1. Fluorine, uniting from a hydroxide apatites enamel, substituting for ОН – groups, forms a fluorapatites, doing an enamel more strong and more proof to the action of acids. This connection reduces permeability of enamel.

2. The mechanism of anticarious action of fluorides is related also to their oppressive influence on growth and exchange of matters of microflora of cavity of mouth.

3. Connections of fluorine in saliva inhibit the transport of glucose in the cages of pathogenic bacteria and formations of for cellular polysaccharidess, which form the matrix of dental deposit.

4. Fluorides violates absorption of microorganisms on-the-spot cages of tooth, absorb albumins of saliva, glycoproteins, as a result of what prevent growth of dental name-plate.

5. And finally, at internal introduction the fluorides normalize an albuminous and mineral exchange.

Fluorides present in enamel and in the dental deposit catalysis «proceeding» in the early carious defeats due to remіneralіzation of crystals of enamel, multiplies the size of crystals of hydroxide apatites.

From modern international data days’ even receipts of fluorides are distributed thus:

–    very low = 0,1-0,6 milligrams;

–   low =0,7-1,4 milligram;

–    optimum = 1,5-4,0 milligrams;

–    high (impertinent fluorosis) = 5-12 milligrams;

–    ever-higher = 20 milligrams and more (at treatment of osteoporosis of bones by fluorines preparations).

The amount of fluorine in an organism depends on his maintenance in water and food products.

ENDOGENOUS PROPHYLAXIS

A specific endogenous prophylaxis provides for:

1. Fluorination of drinking-water

2. Fluorination milk

3. Fluorination salts

4. Adopting the pills of fluorides of sodium

FLUORINATION OF WATER

One of the acknowledged methods of prophylaxis of caries there is fluorination of drinking-water   is   controlled  addition  of connections  to the fluorine  to   water of sources of water-supply with the purpose of leading to the concentration of ions of fluorine in        a drinking-water    to    the level,    which    is   sufficient    for    the effective prophylaxis of teeth decay and at the same time does not have an unfavorable influence on functional possibilities of organism of man, physical development and health of population.

First artificial enriching of tap water by fluorine it is carried out in 1945 and since got distribution more than in 35 countries in which over 150 million persons now    use    fluorination water.

For artificial fluorination to water add the followings connections to the fluorine: fluoride of sodium. This process is carried out to the step waterworks. It is rotined that for achieving maximal efficiency fluorination water, it follows to consume from birth, but some researches rotined efficiency of this method of prophylaxis and at patients which got the optimum concentrations of fluorides upon termination of odontosis.

Presently about 5% all population of earth (approximately 260 million persons) drink fluorination water. In spite of numerous objections of opponents of fluorination, the presence of undesirable effects is not well-proven, and although every objection must be explored, safety of fluorination of water can be considered set.

The optimum concentration of fluorine in a drinking-water is 1,0 mg/l. Expedience of fluorination of water in every case is set by the organs of sanitary-epidemiology service. To fluorination of water low KF” is a testimony in water and considerable staggered of population by the caries of teeth. In theory at any KF”, less than optimum, it is expedient fluorination water, but practically in the first turn it needs to be done on those plumbings in which water contains less after 0,3…0,5 mg/l to the fluorine.

Fluorination of water remains the advantageous and cheap method of prophylaxis. Most influencing of fluorination water shows up on smooth surfaces, in more small degrees on proximal, cheeks and figures. For frontal teeth, influencing of fluorinating water shows up in more small degrees that for masticatory.

Fluorination of drinking-water allows get reduction of increase of baby teeth permanent decay on 40-50% — on 50-60%.

Efficiency of fluorination is estimated by the dynamic supervision during 10—15 years after morbidity by the teeth caries of population which uses fluorinating water. It is possible also to compare findings to the indexes of morbidity by the caries of population of neighbouring settlement which uses water with small maintenance of fluorine (more small after 0,2 mg/l). For the estimation of anticarious action of fluorine use two indexes: prevalence of caries and his intensity (CSR and cs). About satisfactory efficiency it is possible to talk in that case, when in 7—8 years after the beginning of fluorination of water the index of CSR of children 7—8 years went down on 40—50%, about good — on 50—60%, about excellent — on 65—80%.

 

FLUORINATION OF MILK

The use of fluorinating milk is the alternative and effective method of prophylaxis.

Milk a long ago brings over to itself attention of researchers on a number of reasons, so as:

–    it is the necessary component of feed of child, especially in the first years of life;

–    owns valuable nourishing properties necessary for child’s organism;

–    it is the basic source of calcium and phosphorus, fabrics of bones and teeth necessary for the structure;

–    contains the high level of calcium, phosphorus and lactose which laminates carbonhydratess also.

Similar composition allows to milk to bring in the payment in the process of remіneralіzation of enamel of teeth and in its defence.

For successful introduction of method of fluorination of milk certain terms are needed:

–    high dental morbidity of population is in a region;

–    low maintenance of fluorides is in a drinking-water;

–    absence of other sources of system receipt of fluorides.

Except for it, at introduction of similar project striving to is the necessary mortgage of success collaborate from the side of regional administration and organs of health protection, as necessary refinancing for the production and organizational measures at the division and delivery of milk in the organized collectives.

For fluorination of milk more frequent fluoride of sodium is used. The table of contents of fluoride in milk concernes for help fluoride of selective electrode. Technology of fluorination of milk is simple and does not present difficulties.

The amount of fluoride, which must be added to milk, is guilty to take into account age of child and receipt of fluoride from other products and water. Yes, coming recommendations of WOHP from, for children from 3 to 7 years day’s receipt of fluoride makes 0,87-1,75 milligrams.

For the children of preschool age the concentration of fluoride in milk, even 2,5 mg/l, is optimum, as 1,0-1,15 milligrams of fluoride provide the daily total receipt in days.

At the use of fluorinating milk it is necessary to adhere to the followings recommendations:

–    effectively to use this method at children from 3 to 12 years

–    daily a child must use a 1 glass of milk from 0,5 mg of fluoride

–    during a year a child must drink milk not less 250 days.

 

ADOPTING PILLS OF FLUORIDE OF SODIUM

 

The doses of fluoride of sodium are recommended in pills

 

AGE

AMOUNT OF PILLS ON DAYS

FLUORINE, MG

2-4

0,5

0,25

5-6

1

0,5

7-14

2

1

 

 

Pills are effective during development and ripening of teeth. Using it is needed 200-250 days in a year from 2 to 15 years. In this case effect from their use it is possible to compare to influencing of fluorinating water. It is the best to accept in the morning and chew pills. It is thus provided as general so local effect of fluorine. Adopting the pills of fluorine it is possible to be on duty with adopting the drops of Vitaftorum.

  Pills are appointed daily to 14-15-years-old age. Contra-indication to setting of pills:

– table of contents of fluorine in an environment more than 50% from optimum;

– any another ways of adopting a fluorine inward.

By major advantage of adopting the pills of fluoride of sodium inward there is «flexibility» of method at the prophylaxis of teeth caries, that allows to bring a fluorine exactly into those periods, when this more expedient in all, and also exactly to measure out a microelement taking into account age and features of organism. However much this way has failings: difficulty of organization of adopting pills, and, in addition, he appeared more dearst than other methods of bringing fluorine are into an organism.

Experience of the use of pills of fluorides of sodium rotined that only high responsibility of parents, pills constantly tracker after the regular reception by children, can provide a high enough prophylactic effect.

Vitaftorum (Vitaftorum) is the combined fluorine containing preparation. Contains in the composition of fluoride of sodium, vitamins A,D,C.

Pharmacological properties of Vitaftorum are conditioned by combination in him of vitamins A also D2 with a fluorine which favourably influences on the maintainance and forming of fabrics of teeth. Vitamin A assists to the normal odontogeny and correct forming of skeleton. Vitamin regulates the exchange of phosphorus and calcium in an organism, is instrumental in suction them in an intestine and timely laying in a new formed bone.A fluorine finds out the anticarious action, is well sucked in, accumulates in bones, in teeth, by a less measure — in cartilages. Vitamin limits the deposit of fluorine in fabrics and the same prevents intoxication by fluorine.

It is used under time or after-meal during a month inward with an interval in 2-4 weeks each 3 months in locality, where maintenance of fluorine in a drinking-water is minimum. To the children from 1 year to 6 years preparation is appointed for ? of tea-spoon 1 time per a day; from 7 to 14 years — for 1 tea-spoon 1 time per a day during a month. After a 2-a week interruption a course is repeated. In a year 4-6 courses of prophylaxis are conducted with an interruption on summer months.

 

Not specific endogenous prophylaxis provides for:

1. The reception of the calcium of containing preparations

2. The reception of vitamins

3. The reception of roborants.

CALCIUM CONTAINING PREPARATIONS

Calcemin is adopted on a chart, depending on age of child. It is recommended to the use to pregnant, women in the period of lactation, to the children from 5 to 12 years of 1 pill 1 time per a day, after 12 years -1 pill on a day.

Calcium D3 Nicomed – is accepted for 1 pill 2 times per a day in the second half of day – 10 days; 20 days – for 1 pill in a day in the evening. On the average course of treatment by preparations of calcium of makes 1month.

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In a mouth liquid the use of preparation of CALCINOVA which contains a calcium and phosphates necessary for mіneralіzation of bones and teeth, and also complex of vitamins, including the vitamin of D3, which is instrumental in absorption of these minerals in the organs of digestion and their division in the organs of digestion, is the alternative method of increase of maintenance of calcium and phosphorus. Preparation contains a calcium, phosphorus, vitamins A, D, B6, C. In connection with that pills it is needed well to chew, in a mouth liquid the considerable increase passes the concentrations of minerals, which promote mіneralіzation and remіneralіzation of teeth.

VITAMINS PREPARATIONS

Videcholum  is preparation of vitamin of D3. It is appointed 1 time per a year in a winter period.

Vitrum – calcium – for children after 12 years for 1 pill in a day during 1-2 months; to the adults – 1-2 pills in days 3-6 months.

ROBORANTS (ADAPTOGENS)

Adaptogens are preparations, mainly vegetable origin, what have general stimulant operating on the basic functions of the system and promote resistance of body to the unfavorable actions.

Ascorbic acid – from 7 to 13 years – for 250 milligrams, from 14 to 17 years – for 750 milligrams of ? 1 time per a day during three days, 1 time on year.

Vitamin Е- to pregnant on 7-10 and 30-32 weeks pregnancies – on a 1 capsule (0,1) or on a 1 tea-spoon 2 times per a day, during 2-3 weeks.

 EXOGENOUS PROPHYLAXIS

It follows to take to facilities of exogenous prophylaxis:

–    fluorine containing facilities for local application

–    remineralization solutions

–    encapsulants

FLUORINE CONTANING FACILITIES

 

At the use of fluorine it is needed to take into account the following:

1. The concentration of fluorides not must exceed for a local prophylaxis
1-2% (calculating on a fluorine), as with the increase of concentration efficiency 
does not grow.

2. Efficiency of influencing is conditioned by their concentration in the free ionized kind.

 3. It is necessary to take into account in this connection fastening possibility fluoride ions with the ions of calcium.

4. Fluorides is appointed taking into account maintenance of fluorine in a drinking-water and climatic factors.

 

Tooth-pastes

Presently became obviously, that the decline of morbidity by caries is explained to the wide uses of fluorine containing of tooth-pastes.

Pastes are given recommended to the use from 3-4 years. Cleaning is necessary 2 times per a day for 3 minutes, consistently clearing all surfaces of tooth.

Fluorine containing varnishes

One of widespread facilities of local prophylaxis there are varnishes which use for the prolonged period of influencing of fluoride on enamel. They form tape adjoining to the enamel, and which remains on teeth during a few hours and in fissures a few days and even weeks.

A fluorine – varnish shows itself composition of natural resins of vegetable origin. At the market presented: „Ftorlac” (Kharkiv), varnish „Duraphat”, „Belac” (Vladmiva).

A method is given recommended at the moderate or high level of intensity of caries of teeth, to the children and young people with the high risk of origin of caries. Frequency of causing of varnish is 2-4 times per a year, depending on activity of caries.

 

BELAK – F

 

Method: the surface of teeth is purged from the deposit and is dried out. Then by the special brush varnish is inflicted by a skim on the surface of tooth. At the same time it is possible to cover all teeth on one jaw or 3-5 teeth. For getting dry of varnish it is needed about 2-3 minutes. It is possible to dry the varnish by the compressed air. After coverage of teeth by fluorine varnish it is impossible to use the meal of 1-3 hours and in future the desired only spoon-meat. It is not recommended to clean teeth 24 hours. Varnish is contained on-the-spot tooth not less 12 hours and for this time his ions penetrate on a depth to 100 mcm of healthy enamel.

To cover teeth by varnish it follows depending on activity of cariosity: at a 1 degree — 2 times per a year, at 2 — 4 times per a year, at 3 — from 6 to 12 times per a year. Triple coverage of teeth is recommended with an interval 1-2 days.

It is set that in a year after application of fluorine containing varnish second caries of teeth goes down on the average on 50%.

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Fluorine containing gels and solutions for the professional use

Gels and solutions of fluoride of sodium 1% and 2% are used for appliques and electrophoresis. A doctor-dentist conducts procedure in the conditions of policlinic. Remineralization action of gels is based on diffusion of matters from gel in saliva and from her in the enamel of tooth.

 

BELAGEL – F

 

Method: teeth preliminary clear, dry out and impose the wadding tampon well moistened by solution of fluoride of sodium on 3- 5 minutes. At first assess the masticatory surfaces of teeth, and then – labial and cheeks on both jaws. If gel is used, he is inflicted by heated by a brush and give to dry out. After procedure does not recommend eat and drink during 2 hours.

As a rule conduct 3-5 appliques by solution twice on a year and 2-6 appliques by gel on a year.

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Causing of fluorides by a spoon:

1. To choose the spoon of the proper size

It must be the covered is all dental row, including the areas of retraction and it follows to provide access of gel to the contact with the structure of teeth. The ends of spoon (peripheral areas) must be closed in order that gel did not flow down in the cavity of mouth sick. Ideally spoons befit with coverage from the made foam material, as they fit snugly dental row of patient and allow to gel to achieve all surfaces

2. To place gel in a stretcher.

3. To insert a spoon in the mouth of patient.

4. To insert between the spoons of saliva ejector, making sure, that to the patient comfortably (at this method for balance of bite from opposite sides necessary wadding rollers)

5. To bring a spoon out of mouth sick.

6. To ask a patient to spit out immediately after the delete of spoon.

After procedure, at a necessity the delete of superfluous fluoride, to apply the intensive sucking

7. To warn a patient that during 30 minutes after procedure it is impossible to eat or drink

Fluorine containing solutions for the independent use

The wide use in the prophylaxis of caries was found by solutions with the low concentrations of fluoride.

The rinses begin to use, when the first second teeth cut through at children. The method of prophylaxis is given does not need considerable expenses of time and financial resources, and that time is effective enough.

Amount of rinses makes:

– by a 0,05% solution -1 once on a day

– by a 0,1% solution -1 once in a week

– by a 0,2% solution – 1 one time in two weeks

For the improvement of co-operation of fluorine with an enamel preliminary it follows well to clean teeth and rinse a mouth by alkaline water for the change of рН environment. In addition it is necessary to teach a child to rinse a mouth by ordinary water by energetic motions by cheeks.

Method: depending on age of child give to rinse the mouth of 5-10 ml solution. The rinse lasts 1-3 minutes, and for the children of junior age more expedient double rinse on 1 minute. After it a mouth is rinsed by clean water.

Application by the children of rinses by solution of fluorine sodium gives reduction to the caries 30%, upon termination of rinses an effect lasts yet in 2-3.

At casual swallowing of fluoride of sodium it is necessary to give to the child to take an a swig at the soupspoon of solution of chloride of calcium, which, linking a fluorine will not give him to be sucked in.

Application of fluorine containing disks

Fluorine containing disks (paper and paraffins) are produced in packing for 10 things. The expense of material is a 1 disk on procedure. The disk of «Ftorglicofoskal’» contains the followings ingredients:

– neurosin — 8-16 g

– fluoride of sodium — 0,5-2 g

– superficial matters — 0,5-2,0 g

– beeswax — 4,5-6,5 g

– paraffin.

A disk is fixed in an angular tip by a mandrel. A fluorine is rubbed in hard fabrics of tooth on minimum speed with the use of three types of motions: recurrently-forward, up-down, circles.

As usual, before treatment by fluorine containing disks the professional hygiene of cavity of mouth is conducted, whereupon by disks at first the vestibular surfaces of all teeth of maxilla are processed from left to right, then lower — from right to left. After it the palatal surfaces of teeth of maxilla and languages surfaces of teeth of lower jaw are processed, farther are masticatory surfaces of teeth of overhead and lower jaws with the use of only circular motions clockwise. At a variable bite process by fluorine containing disk only the second teeth. It is recommended 2-3 multiple treatment of teeth with an interval 1-2 days, in a year 2-4 courses.

In practice of therapeutic dentistry fluorine containing disks found large popularity not only at the prophylaxis of teeth decay but also at treatment of hyperesthesia of hard fabrics of teeth.

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Remineralization solutions

At application of remineralization matters it is expedient to heat them to 40-450C, taking into account that the increase of temperature of solution on 10 strengthens precipitation of ions on-the-spot enamel on 1 %.

Choosing the concentration of remineralization solution, it should be remembered that the high concentration of calcium conduces only to mіneralіzation of superficial layer of enamel, while the low concentrated solutions are instrumental in remineralization on all depth of enamel.

In behalf of the combined application of remineralization solutions and solutions of fluorine the fact of assistance of ions to the fluorine testifies to the acceleration of plugging in the net of enamel of calcium and phosphorus.

 

Borovskij’s – Leus’s method

Teeth carefully purge from the dental raid by ordinary pastes, dry out.

Consistently conduct appliques by a 10% solution of glucoside of calcium during 15 minutes(3 times for 5 minutes) and 2% by water solution of fluoride of sodium during 3 minutes. Duration of course on the average 10-15 procedures every day or in a day.

 Borovskij’s – Leus’s method

(with electrophoresis)

This chart foresees electrophoresis of a 10% solution of glucoside of calcium (3—5 minutes) and applique of a 2% soluble- fluoride of sodium (1—2 minutes) 3 times in a week.

 

Borovskij’s – Volkov’s method

Two-component solution which consists of a 10% solution of nitrate of calcium and 10% solution of hydrophosphate to the ammonium is used. Prepare teeth and consistently conduct appliques by each of these solutions for 3-5 minutes. Through 5-7 procedures on-the-spot enamel and in mіcro spaces under superficial layer the matter appear which is an ionogen phosphorus and calcium. This method is used for treatment of hyperesthesia.

 

Udovits’ka’s method

O. Udovits’ka recommends to apply at the children of 2—3 years of applique of a 10% solution of glucoside of calcium (minutes) and fluorine varnish 6 times per a week; course — 1 one time in a year during 3 years

 

Applique by solution „Remodent”

Remodent”  is the preparation synthesized from natural materials consists of complex of ions macro- and micrtoelements necessary for activating of process of remіneralіzation and prophylaxis of caries. Unlike fluorine preparation is instrumental in substituting for the ions of calcium and phosphorus in the crystalline grate of enamel of teeth.

On preliminary cleared (by a tooth brush and tooth-paste) and dried up teeth inflict the wadding tampon saturated by solution of “Remodent”    on 15—20 minutes. During this time a tampon is changed twice. After an applique it is not recommended to rinse a mouth and adopt a meal during 2 hours. Next appliques conduct twice for a week after the same method. The course of treatment is 20—30 appliques.

In addition, with the purpose of prophylaxis of teeth decay it is possible to recommend the rinse of mouth by a 3% solution of “Remodent” by duration of 3—4 of minutes (1—2 times per a week) during 10 months on a year. On one rinse goes to 15—25 ml solution.

 

 Vinogradova’s method

Purge teeth by hygienical tooth-paste, appliques dry out and conduct teeth by a 10% solution of glucoside of calcium on 2-4 minutes and mouths baths (washing) by a 0,2% solution of fluoride of sodium on 2-4 minutes. This complex is conducted 3-4 times per a year.

In the over concentrations fluorides is toxic.

 

 Recommendations are on the hygienical care of cavity of mouth of children

Parents must begin the hygienical care of cavity of mouth of child from the moment of eruption of the first temporal tooth (in age 5-6 months).

This procedure needs to be executed 1 time per a day (in the evening before sleep). For the removal of dental deposit from every surface of tooth the special very soft tooth brush is recommended, what dressed on afinger. From gums to the cutting edge clear the teeth of child circular motions without application of tooth-paste.

 

To the moment of eruption for a child 8th teeth (as a rule, to one year) it is necessary to clear teeth parents already twice in a day (in the morning and in the evening) by soft child’s tooth brush (length of working part must not exceed 1,5 mm), also without application of some tooth-paste. Thus the special attentioeeds to be spared teeth which are in the stage of eruption the masticatory surfaces of which did not yet attain the level of occlusal plane.

Teeth it is recommended to purge thus:

1. To clear vestibular surfaces at serried jaws, here to set a tooth brush horizontally, athwart to the surface of teeth, carrying out only vertical motions in direction from gums to the cutting edge of teeth: on an overhead jaw – from top to bottom, on lower – from below to upwards .

To clear oral surfaces circular motions, here in the area of frontal teeth on an overhead and lower jaw to set a brush apeak.

It follows to delete a raid from the masticatory surfaces of teeth motions ahead-back.

It is recommended to use this technique in the period of eruption of all temporal teeth (to 2-2,5), when cleaning of teeth must become for a child by obligatory part of morning and evening rest room.

In 2-2,5 parents must clean teeth children by soft child’s brush twice on a day (in the morning – to breakfast and in the evening – before sleep) and to use child’s gel tooth-paste.

 

To prevent tooth decay:

·                     Brush your teeth at least twice a day with afluoride-containing toothpaste

. Preferably, brush after each meal and especially before going to bed.

·                     Clean between your teeth daily with dental floss or interdental cleaners, such as the Oral-B Interdental Brush, Reach Stim-U-Dent, or Sulcabrush.

·                     Eat nutritious and balanced meals and limit snacks. Avoid carbohydrates such as candy, pretzels and chips, which can remain on the tooth surface. If sticky foods are eaten, brush your teeth soon afterwards.

·                     Check with your dentist about use of supplemental fluoride, which strengthens your teeth.

·                     Ask your dentist about dental sealants (a plastic protective coating) applied to the chewing surfaces of your back teeth (molars) to protect them from decay.

·                     Drink fluoridated water. At least a pint of fluoridated water each day is needed to protect children from tooth decay.

·                     Visit your dentist regularly for professional cleanings and oral exam.

C:\Users\Katya\Desktop\images (3).jpg

 

 

Caries of deciduous teeth in children. Patterns of clinical manifestations and course. Diagnosis, differential diagnosis.

Caries of permanent teeth in children. Patterns of clinical manifestations and course. Diagnosis, differential diagnosis.

Dental caries (caries dentis) – a pathological process that develops after teething and is characterized by demineralization and destruction of hard tooth tissue with subsequent formation of a defect in a cavity. According to the WHO nomenclature for the assessment of staggered teeth caries using three basic indicators: prevalence, intensity and increase the intensity of caries.

The prevalence of caries – is the ratio of people ( as a percentage ) that are carious , sealed and removed teeth, ¬ tion to the total number of patients. The prevalence of caries in different ages varies vschobrazhena This pattern of caries in the age curve (Fig. 25). Carious lesions are diagnosed in children aged 1-1.5 years . At the age of 7-9 years, the prevalence of caries increases to 95-100% in children 11-12 years decreases to 70-80 % due to physio ¬ logical change of teeth and the formation of permanent occlusion . Since 13-14, the prevalence of caries years growing again.

         Intensity decay (caries index) – the number of carious teeth removed and sealed in a single surveys. In children with tymcha sovym bite caries index referred to as “CP” (carious, seals ¬ ated) as deleted temporary teeth to be recorded. In children with varying dental occlusion index referred to as CPV + kp (take into account yuhsya affected by caries as temporary and permanent teeth). In children with permanent occlusion of caries index is designated as CPV (carious, sealed, removed).

 

Мал. 25. Вікові періоди високого та низького приросту карієсу зубів

 

 

         Таблиця 18. Оцінка інтенсивності карієсу зубів за віком (за умови зниженого вмісту фтору в питній воді)

Age, years

The level of caries

Very High 

 High

Low

 Resistant to decay

2

3 and more

2

0

0

3

6

2-5

1

0

4

9

3-8

2

0

5

9 more kp 1KPV

4-8

1-3

 

 

7

10 more kp

7-9 CP 1-4 CPV

1-6 СP

0

9

10 and more 5 and kp

CPV more

6-9 kp3-4 CPV

0-5 kp 0-2 Cpv

0

11

8 CPV and more

5-7 CPV

1-4CPV

0

13

12 and more

6-11

1-5

0

15

15 and more

9-14

3-8

0-2

17

18 and more

9-17

4-8

0-3

19

21-32

14-20

7-13

0-6

20-24

26-32

18-25

9-17

0-8

25-29

28-32

20-27

12-19

0-11

30-34

28-32

20-27

13-19

0-12

35-39

29-32

22-28

14-21

0-13

40-44

32

22-32

14-21

0-13

 

As an indicator of the intensity of dental caries , along with an index that takes into account the affected teeth removed and applied index, which takes into account the number of caries -affected surfaces of teeth – Cpt. To determine the index counts the number affected by caries or tooth surfaces sealed . This index more accurately reflects the dynamics of the caries process , which is especially important in determining the effectiveness of preventive measures.

For evaluation of these processes ( dynamics caries development and its effective prevention) used indicator of intensity of growth – decay. To determine the intensity of the su ¬ Hazel ( the index of dental caries or tooth decay index surfaces) in the same person or contingent after a certain period ( 1-3-5 years). The difference in value of the index between the second and the first survey is increase the intensity decay.

 

 

Etiology

To explain the etiology and pathogenesis of dental caries suggested about 400 theories, the most famous of which contributed to the accumulation of information that allow to express a complete view of the problem.

Without going into detail on all existing theories are given below that at least to some extent, give an explanation of the origin of the most common pathological process – caries.

Theories of the origin of dental caries

1. Chemical-parasitic theory of caries Miller (1884). At one time this teoriyab Ula progressive, was recognized and fairly widespread. Nowadays, the theory of tooth decay was the basis for the modern concept of the pathogenesis of dental caries. According to this theory, the carious destruction occurs two stages:

a) demineralization of dental hard tissues . Lactic acid is formed in the mouth , resulting in lactic acid fermentation of carbohydrate food residue dissolves inorganic substances enamel and dentin ;

b) is the destruction of organic matter dentin proteolytic enzymes of microorganisms.

However, factors such as microorganisms and acids , Miller acknowledged the existence of predisposing factors . He pointed out the role of the quantity and quality of saliva, factor supply, drinking water, emphasized the importance of hereditary factors and conditions of formation of enamel.

2. Physico-chemical theory of caries DA Entin (1928). Entin proposed the theory of decay obtained from the physico- chemical properties of saliva and tooth. He believed that the tooth is semi-permeable membrane through which the osmotic currents caused by the difference in osmotic pressure between two media in contact with the tooth , blood and saliva from the inside out. According to him, under favorable conditions, osmotic currents are centrifugal direction and ensure the normal supply conditions dentin and enamel and prevent action on the enamel of unfavorable factors. In turn, the centripetal movement of substances , ie the surface of the tooth to the pulp was considered abnormal and little direct connection with diseases of the nervous and endocrine systems, heredity , disturbance of mineral metabolism , conditions harchevannya , life , work, physiological relationships that lead to violations pulp in the system – tooth – saliva. Under adverse conditions centrifugal direction osmotic currents weakened and becomes centripetal direction that gives the power of enamel and facilitates action it harmful external agents ( microorganisms) , causing cavities.

Disadvantages theory Entin did not know that at the centripetal flow of substances in enamel occurs nutrition enamel minerals from saliva , and this method is based exogenous caries prevention – covering ftorlaka teeth , causing remineralyzuyuchyh substances ( applications) on the surface of the enamel with a view to refill their enamel – remterapiya , as well as methods of treatment of caries in stage spots : application to the affected area of enamel remineralizuyuchyh substances. 

image002

The scheme of the pathogenesis of dental caries by DA Entin

 

3. The biological theory of caries IG Lukomsky (1948). The author of this theory believed that endogenous factors such as lack of vitamins D, B, and inadequate and poor value for calcium , phosphorus, fluoride in the diet , lack of or insufficient ultraviolet rays break the mineral and protein metabolism. The result is disease odontoblasts that first weakened and then are inferior. Reduces the size and number of odontoblasts , leading to metabolic disturbances in the enamel and dentin . First comes dyskaltsynatsiya , then a change in the composition of organic matter. The theory has no experimental evidence. The negative aspects of this theory: there is no evidence that the odontoblasts are trophic centers of the tooth , theory does not explain the role of sugar in the development of caries, carious lesions and localization preventive effect of fluoride , not proven to be defective in odontoblasts caries . Even in a healthy tooth contact can be found degeneration of odontoblasts in the form of vacuolization and atrophy.

4. Theory of AE Sharpenaka (1949). Sharpenak explained the cause of tooth decay tooth enamel local depletion of proteins as a result of their rapid decay and slow resynthesis , which inevitably leads to caries in a stage of white spots. Slowing resynthesis caused by the absence or low content of amino acids such as lysine and arginine, and the reason for enhancement of proteolysis is a high ambient temperature , hyperthyroidism , nervous overstimulation , pregnancy , tuberculosis , pneumonia , accumulation of acids in the tissues of the body (in particular, insufficient flow of vitamin group, tissues accumulate a large amount of pyruvic acid) , which leads to increased protein breakdown. Kariesogennoy effect of carbohydrates Sharpenak explains that when a large of learning increases the need for vitamin B1, which can cause vitamin deficiency and increased proteolysis in solid substances tooth. Disadvantages theory: not confirmed experimentally that when caries in a stage of degeneration begins proteolysis of proteins , author underestimated the role of microorganisms kariesogennoy local factors and overestimated the role of common factors.

5. Proteolysis-decay theory helatsionna Shattsa and Martin (1956 р). The authors explain the perception of enamel to caries stable calcium -protein complexes. Tooth enamel is the integral structure of the body, which is due to the functional features of mineralized than other tissues. Thus the mineral and organic components of the enamel are in close biochemical connection. The stability of the latter may be affected by penetrating the enamel of different active chemical agents including proteolytic . The development of the caries process is considered in two stages:

a) proteolysis in which the break bonds between proteins and minerals enamel

due to the action of proteolytic enzymes on bacterial protein components ;

b) helatsiya when there is destruction of the mineral part of dental hard tissues due to the formation of complex compounds of metal ions with anions of acids, salts of organic acids , amino acids , proteins and intermediates decay.

Disadvantages theory: there is currently no evidence of the first phase of the caries process by Shattsu – Martin . At the stage of caries spots or breach of covalent bonds between organic and inorganic substances , or protein breakdown have been identified. Underestimated local action of acids produced during dental plaque and overrated helatsionnyy method demineralization of enamel, which is very slow, while the destruction of dental hard tissues is sometimes a very intensive. The theory does not explain the localization of caries lesion frequency specific tooth surfaces.

6. Trofonevrotychna Theory EE Plato. By this theory, considered as trofonevrotychnyy caries process, which , in his opinion , develops only when the power supply is broken dental hard tissues. The main pathogenetic factor EE Plato believed trophic disorders of the nervous regulation of dental tissues. But today we know that teeth with pulp removed in time to 17 years continue to functioormally. Removing the pulp as a method of treatment of complicated forms of caries does not lead to structural and functional changes in the enamel of the tooth , the latter continues to function as a full body. Constant dynamic interaction with oral fluid provides hard tissues of the tooth depulpovanoho high mineralizovanist , which corresponds to high acid stability , microhardness and structural homogeneity.

The modern concept of the etiology of dental caries.

Based on historical theories are being made ​​significant progress in the study of the etiology and pathogenesis of dental caries. Generally accepted mechanism of caries is a progressive demineralization of dental hard tissues under the influence of organic acids, the formation of which is connected with the activity of microorganisms. In the event of the caries process involved many etiological factors, allowing polietiolohychnym count caries disease.

The main etiological factors are:

1. Oral cavity;

2. The nature and diet, fluoride content in water;

3. The quantity and quality of saliva;

4. The general condition of the body;

5. Extreme action on the body.

All of the above factors were identified kariesogennoy and subdivided into general and local, such that play an important role in the occurrence of dental caries.

General factors:

1. Inadequate diet and drinking water;

2. Somatic diseases, changes in the functional state of organs and systems during the formation and maturation of tissues.

3. Extreme action on the body;

4. Heredity, causing the usefulness of the structure and chemical composition

tissues. Adverse genetic code.

Local factors:

1. Dental plaque and plaque that is isolated microorganisms;

2. Violation of the composition and properties of oral fluid;

3. Hydrocarbons sticky food residues mouth;

4. Resistance dental tissue caused complete structure and chemical composition of dental hard tissues;

5. Abnormalities in the biochemical composition of dental hard tissues and defective structure of tissues;

6. State dental pulp;

7. State of dentition during bookmarks, development and eruption of permanent teeth.

Kariesogennoy situation is created when any kariesogennoy factor or group acting on the tooth , making it susceptible to acids. Of course, the trigger is the oral cavity with obligatory presence of carbohydrates and touch two factors of tooth structure .

In terms of lowered resistance of dental tissues kariesogennoy situation develops easier and faster.

Clinically the mouth kariesogennoy situation is the following symptoms:

1. Poor oral hygiene;

2. Abundant plaque ;

3. Plaque ;

4. Overcrowding of the teeth and bite abnormalities ;

5. Bleeding gums.

Stability of dental caries or tooth decay resistance provided by:

1. The chemical composition and structure of enamel and other tissues ;

2. The presence pellikuly ;

3. The optimum chemical composition of saliva and myneralizuyuschoyi its activity;

4. Sufficient amount of oral fluid ;

5. A low level of permeability of tooth enamel ;

6. A good chewing load and self-cleaning surfaces of the teeth ;

7. Properties of plaque ;

8. Good oral hygiene ;

9. The features of the diet;

10. Correct formation of primordia and the development of dental tissues ;

11. Timely and complete maturation of enamel after eruption of the tooth;

12. Specific and nonspecific protective factors of the oral cavity .

Susceptibility to caries or tooth karyyesspryymannya contribute:

1. Defective maturation of enamel;

2. A diet deficient proteins , macro -and micronutrients , excess carbohydrates;

3. Water from the lack of fluoride ;

4. Lack pellikuly ;

5. The composition of oral fluid , its concentration , viscosity, amount and rate of discharge;

6. Biochemical composition of hard tissues , which determines the course of caries as a dense structure of spaces with minimal lattice slows the progress of tooth decay and vice versa;

7. State of the neurovascular bundle ;

8. The functional state of organs and systems during the formation and maturation of tissues ;

9. Improper development of the tooth due to common somatic diseases.

In Table. 19 presents some general and local factors charac ¬ Theroux that cause caries in children.

Table 19. Global and local causes of tooth decay in children

 

According to modern concepts , the development of tooth decay occurs vnas ¬ lidok complex interaction of internal and external factors that imple ¬ zuyetsya microorganism in the system – saliva – the structure of the enamel (Fig. 26).

The effect of these factors is easier to figure out if the normal state of enamel seen as a dynamic equilibrium between the constant process of de- and remineralizatsp . In the case when the dental tissues processes Deming – ralizatsiyi outweigh remineralization occurs demi – site neralizatsii as carious spots. Further progression of the process ¬ demiyiyieralizatsiyi enamel and dentin leads to the formation of brown ¬ oznoyi cavity.

It is known that the development of dental caries associated microflora , among which the leading role played by streptococci, especially Str. mutans. Etiological role of microorganisms in the development of dental caries demonstrated in experiments on animals hnotobiotychnyh who have not decay occurred despite prolonged exposure to kariesogennoy diet. Launching kariesogennoy ordinary food microflora of the oral cavity ¬ us human constant led to the development of caries in experimental animals.

Kariesogennoy action of microorganisms associated with the formation of their dental plaque . It is a conglomerate backbone is made up of micro ¬ organisms polysaccharide fixed in the stroma , which to varying degrees in ¬ saturated with minerals . Dental plaque is flat against the tooth surface and is over pelikuloyu – ¬ tion of organic thin film that covers the tooth enamel. Dental plaque is most peo ¬ ple contains the same basic types of microorganisms in different ratios (Table 20). Thus the fate of streptococci (mutans, salivarius, mitis, sangvis) accounts for about 40 % of the total microbial plaque.

 

Table 20. The microflora of dental plaque (by Ch. Moutoun, 1993)

 

         Most kariesogennoy th Str. mutans, which produces only lactic acid from glucose , mannitol and sorbitol splits , forming glycans of sucrose. Loss of ability to produce glycans leads to loss of virulence Str. mutans.

The formation of dental plaque occurs in the sequence:

1) the attachment of bacteria to pelikuly ;

2) the formation zovnishnokli hospitable structure ( matrix );

3) the growth of bacteria and the formation of plaque .

There are various mechanisms of attachment of bacteria to pelikuly . LM.Silverstone ( 1980) points to the existence of such phases in this process :

– Adsorption mikromolekul ;

– Chemical attachment of bacteria cell ;

– Return fixing bacteria on the surface;

– Clinched their fixation ;

– Develop secondary microflora.

Chart. 26. Figure Keyes (1962), which reflects the interaction between the three bases ¬ them etiological factors of dental caries


This issue pay much attention to the fact that fixing bacteria reduction will reduce the potential for kariesogennoy in ¬ upper tooth.

Formation matrix associated with the activities of microorganisms. Matrix consists of two components : proteins, glycoproteins mainly derived from saliva, and bacterial extracellular policy ¬ harydiv (mainly carbohydrate polymers ).

For thickening and ” maturation ” of porous and permeable ¬ f us ” young ” dental plaque creates anaerobic conditions that change ¬ tion of microbial composition . The formation of dental plaque is largely due to the disposal of bacteria food debris trapped on the surfaces of the teeth. Most bacteria use substances that readily penetrate the plaque : sucrose , glucose, fructose, mal ¬ tozu , lactose. Of particular importance nadastsya dextran , because the number of significant ¬ adhesive properties required for the fixation and growth of dental plaque , the poorly soluble polysaccharide ¬ priorities would need to resistant microorganisms.

Glycolysis carbohydrates ( sucrose , glucose, fructose ) contributes lo ¬ radical reduction of the pH on the surface of the tooth enamel to a critical level (pH = 5.0 ), accompanied by an increase in the permeability of enamel (Fig. 27). Prolonged maintaining a critical level of hydrogen ions occurs acid dissolution apahytiv surface in the least stable parts of the enamel ( line Rettsiusa , mizhpryzmovi spaces ), accompanied by the penetration of acids in the subsurface layer of enamel demineralization its next.

 

Chart. 27. The process of acid from the interaction between bacteria and kariesogennoy carbohydrates (sucrose). Prevalence of enamel demineralization processes in case of lack of local mechanisms for neutralizing acids (by Ch.Moutoun,1993)

VK Leont’yev and AI Vershinin (1991) suggest a parallel ¬ ing course

two reactions: hydroxyl uhvoryuyetsya is not 10, and 9 atoms Ca2 ^ one of them is substituted for a hydrogen ion (H ^ or hydronium (HgO ^). hydroxyapatite The structure is not destroyed, but decreases its ability to resist acids due a reduction of calcium ¬ tion and therefore calcium-phosphorus coefficien.

Changes in the surface layer of enamel are less pronounced than in the deeper parts of it, due to its structural features (presence of large amounts of fluorapatite) and reminera-ation processes that occur continuously as a result of receipt of the mineral component of oral fluid. The subsequent formation of organic acids on the surface of enamel demineralization leads to increased and gradually increase mikroprostoriv between crystals of enamel prisms. As a result, the conditions for the penetration of microorganisms and their metabolic products of micro-defects of enamel. The source of acid is transferred within the enamel. At this stage of demineralization of enamel caries process extends both along the surface and at depth, forming a cone-shaped cell destruction. Continued existence of areas of demineralization leads to the dissolution of the surface, more resistant enamel layer to form a defect. Consequently, the initial clinical caries enters surface.

Also kariesogennoy action of dental plaque, are important factors that contribute to resistance or susceptibility to dental caries (transferred and related diseases, malnutrition, etc.., Figure 3). Established that in adults and children who have suffered acute infectious disease or suffer from chronic diseases of internal organs and systems, especially often caries. The degree of infestation caries is not associated with one or other person  portunities transferred or concomitant disease.

For various diseases accompanied by increased susceptibility to caries, general him with the fact that they are characterized by changes in the state of immunological reactivity. These changes are primarily in reducing the number and functional activity of cellular and humoral protective factors in both serum and oral fluid in his Principles humoral factor in local anti-infective resistance of the mucous membrane of the mouth is IgA, specifically secretory (S IgA). They prevent adhesion of microorganisms to the mucosal surface of the oral cavity, as well as the dental hard tissues. In addition, S IgA saliva can alter metabolism of microorganisms to limit the formation of colonies, to reduce the virulence of infectious agents.

The development of dental caries is associated with a decrease in the number or absence of S IgA in saliva. However, this view is not shared by all investigators. The contradictory data on the role of S IgA in the pathogenesis of dental caries due to the fact that the system of secretory antibody labile and depends on many factors, local and general (gender, age, climatic conditions, etc.).

Chronic diseases of internal organs and systems are also accompanied by changes in physical and chemical properties of saliva, decreasing its protective and remineralizuyuchoho potential.

Stability of dental caries is defined as the composition and properties VOST enamel. The structure of the enamel as temporary and permanent teeth conditioned primarily by genetic factors, but largely depends on the usefulness of enamel mineralization processes (as vnutrishnoschelepnoyi and oral). It is known that mineral ation of deciduous teeth occurs almost entirely in intra-shnoutrobnyy period as crucial health of the mother during pregnancy. On the stability of deciduous teeth caries significantly affect toxaemia of pregnancy, carbohydrate metabolism, thyroid disease, digestive tract viral illness like chronic hypoxia and others. In the temporary teeth of children born to mothers with the above pathology during pregnancy, caries faultpeared almost immediately after their eruption.

In odontohenez deciduous teeth, and hence on the structure of enamel influenced by factors such as prematurity pregnancy, illnessnewly born and children 1 year of life. Results polarization opcal and X-ray analyzes of deciduous teeth indicate that at physiological pregnancy occurs povnotsin ¬ on mineralization of enamel. In toxemia of pregnancy (nephropathy, sudyanka, preeclampsia, etc.). Enamel of primary teeth revealed some areas of defective mineralization. These sites are stored in infancy, even in intact temporary teeth as hipomineralizovanyh zones.

In mineralization of permanent teeth in children than the mother’s health significantly affects the health status of the child, as this process occurs mainly in the first years of life. The nature and adequacy of nutrition, concomitant diseases, functional status herbstion tract content of fluoride in drinking water and other factors may play a role in the formation of caries-resistant or susceptible to dental hard tissues of the teeth.

Established (V.R.Okushko and snivavt., 1989) that the solubility of tooth enamel depends on the functional state of the pulp, which is associated with changes in resistance of teeth to caries. These properties of the enamel as its major strength and low solubility are provided only for maintaining high functional activity of the pulp. Mozh  cially that differences in functional status iiulpy different etagroin of temporary and permanent teeth determine the features of the development and progress of caries in children.

The functional activity of the pulp is largely related to the physical condition of the child. Chronic diseases of internal organs and body systems can lead to a decrease in the functional activity of the pulp, thereby increasing susceptibility to caries and enamel causing aggressiveness of its course.

Saliva creates an environment in which the teeth after eruption are constant.

Saliva performs many functions: it is involved in the processes of self-moochyschennya mouth and teeth has a buffer capacity, itso helps to normalize the pH in the mouth when you reduce it, is supersaturated solution on the content of calcium and phosphorus, as well as a source of receipt of these and other ions to the enamel, ensuring processes of mineralization and remineralization.

Stability of dental caries is directly related to the composition and properties of saliva. Individuals susceptible to tooth decay, the decrease rate of saliva, variations in mineral composition and reduction of antibacterial properties. Similar changes are found in the saliva of pathological conditions, minors notsinnomu food and otherwise, when the teeth are exposed to intense shock syvnomu caries.

Pathogenesis

Local factors kariesogennoy

The direct damaging effect on hard tissues of the teeth have a local kariesogennoy factors. Most kariesogennoy value (either alone or in combination with other factors) have dental plaque: plaque and dental plaque.

Plaque – a yellowish or grayish-white soft gummy deposits on the tooth surface, which is a conglomerate of microorganisms, desquamated epithelial cells, leukocytes, a mixture of saliva proteins and lipids with slices of food. Soft plaque is not constant internal structure inherent in dental plaque, it is loosely attached to the tooth surface, so it can be washed off easily enough running water or remove with a cotton ball. Dental plaque is a soft amorphous granular deposition, which is very tightly attached to the tooth surface, from which the plaque can be separated only by mechanical cleaning instrument or a toothbrush. When dental plaque is small, it is not noticeable (if not painted food pigments or dyes) in case of increasing size, it becomes gray or yellow-gray mass on the surface of the tooth.

In the attachment of dental plaque to the tooth surface is of great importance pelikula.

It is formed after the eruption of the tooth, as it is called acquired cuticle. Pelikula is a fine film that is derived protein-carbohydrate complexes saliva – mucin, glycoproteins, sialoproteyiniv (SD Hogg, I. Lightfoot, 1988). It includes three layers: surface and 2 attached quite firmly to the surface of the enamel. It is believed that pelikula is a kind of connective substance between dental plaque and surface enamel. Prerequisite formation of plaque and dental plaque is the presence of microorganisms. It has been experimentally shown that in animals hnotobiontiv (grown in special conditions of the animal, the body which no bacteria) never formed plaque and plaque. Numerous experimental studies it was found that the formation of dental plaque can be divided into several stages (IL Hardwick, 1985).

And the stage. Formation of cell-free organic film on the surface of the tooth enamel, which is called pelikuloyu or acquired cuticle. Of course it is necessary for the formation pelikuly from several minutes to several hours.

Second stage. On the surface adsorption of proteins occurs pelikuly microorganisms and epithelial cells. They stick to pelikuly gradually started growing bacterial colonies. On average, this stage lasts a few days.

III stage – the creation and formation of mature dental plaque. It happens Precipitation extracellular polysaccharides formed by microorganisms from plaque glycoproteins of saliva. At this stage, the plaque is the greatest threat to tooth enamel as actively secretes organic acids (lactic, acetic, etc..) And hydrolytic enzymes (proteases, hyaluronidase, etc..). Plaque formation begins with the accession of a monolayer of bacteria to the acquired pelikuly or tooth surface. Dental plaque is composed primarily of microorganisms (70%) and extracellular matrix (complex glycosaminoglycans and proteins). If the conditions of dental plaque increases rapidly and reaches a maximum of about 30 days.

Mature dental plaque often has the following structure (Figure.):

1. Acquired pelikula, which provides a link with enamel plaque;

2. Palisadnykopodibno located fibrous layer of microorganisms that settle on pelikuli;

3. Dense fibrous mesh of microorganisms, which are colonies of other types of bacteria;

4. surface layer of microorganisms kokopodibnyh (Z. Broucal, J. Svejda, 1973).

image004

Scheme of the structure nad’yasennoyi dental plaque:

1 – pelikula;

2 – palisadnykopodibno are microorganisms;

C – fiber microorganisms;

4 – coca;

5 – tooth enamel

G.H. Bowden (1985) divides dental plaque microorganisms into 2 groups:

1. Acidophilic bacteria that are able to grow in an acidic environment and ferment acid;

2. Proteolytic bacteria produce proteases.

The first group includes lactic streptococci, lactobacilli, actinomycetes, leptotryhiyi and Corynebacterium. The second group are anaerobes that processed food proteins and amino acids. More important is the separation of microorganisms in dental plaque:

1. Kyslototvorni – ferment carbohydrates to form acids;

2. Microorganisms that ferment carbohydrates to form polysaccharides: dextran, Levante, etc.

The latter form network structure of dental plaque. Up to 50% of the bacterial flora of plaque streptococci constitute acidophilus dominated Str. Mutans and Str. sanguis (II Oleinik et al., 1983, 1986). On the number of species and the number of bacteria affects the availability of substrates in the mouth of food (carbohydrates, amino acids, etc..). For example, in the case of food that contains a lot of sucrose, the number of dental plaque and prevail acidophilus streptococcus.

The main proteolytic bacteria are rystely, which account for over 30% of this group of microorganisms. Important role in the formation of dental plaque play carbohydrates. Established in patients who consume a lot of carbohydrates from food (especially sucrose), dental plaque formed relatively quickly and in large quantities.

Numerous studies have shown that plaque accumulation resulting in him microorganisms able to quickly and easily convert carbohydrates into acids (lactic, acetic, propionic). Acid formation in plaque (plaque) and decreased with pH occurs only in the presence of oral carbohydrates. The easiest way plaque bacteria ferment sucrose, glucose and fructose.

Cellular elements plaque with adhesive protein telementamy ensure its porous structure that is able to pass through a oral fluid (saliva, liquid food). Formed by microorganisms extracellular polysaccharide intercellular spaces in close plaque than contribute to the accumulation of organic acids in it. Thus, plaque and especially dental plaque is a semipermeable membrane that can selectively skip through a variety of substances. Quite easily diffuse into dental plaque carbohydrates that are found there in quantities directly proportional to their concentration in saliva and exposure time. Simultaneously plaque does not pass a saliva alkaline substances that are able to neutralize the acid. Mucoid plaque film is insoluble in many chemicals. In the absence of carbohydrates plaque pH generally ranges from close to neutral – 7.0. The consumption of carbohydrates causes a sharp increase in the acidity of plaque, which lasts about 30 minutes, reaching a pH of 5.8 – 4.5. After this acidic environment is neutralized plaque buffer systems saliva pH is neutral over its values. If carbohydrate intake is repeated, there is a fairly steady decline in pH during dental plaque, causing damage to the enamel. Dissolution of enamel begins when the pH drops to 5.5, so this pH deem critical. When plaque may be a significant increase in acidity. In addition, plaque prevents the penetration of the enamel to saliva alkaline compounds that are able to neutralize acids, and inorganic substances that are constantly coming with saliva in the enamel, restoring its mineral structure.

Thus, under appropriate conditions under dental plaque may form and long maintained a condition where the concentration of organic acids will dissolve the enamel mineral structure, proteolytic enzymes and microbial plaque – split its organic components. The state of the reaction medium during dental plaque and is also influenced by factors such as age, plaque localization (by contact or vestibular surface of the tooth), the presence and concentration of carbohydrates in the mouth, the ability of carbohydrates to diffusion in plaque, saliva buffer capacity. That dental plaque largely gives the functional balance between hard tissues (enamel) and oral, because there is considerable kariesogennoy factor. Dental plaque is liable to cause a variety of immunological reactions. Proved that it contains immunoglobulin A, G, M, amylase, lysozyme, albumin and other protein substrates. Immunofluorescence methods were found to be harboring immunoglobulin tooth plaque and bacteria. For example, bacteria coated with IgA, coming from saliva or gingival fluid (GD Ovruchskyi, VK Leont’ev, 1986; Wiltont IM et al., 1988). This immunoglobulin is the main factor that prevents the adhesion of microorganisms to the surface of the dental hard tissues (enamel) and the formation of their colonies. Given the significant role of antibodies in the development of many pathological processes have attempted to link to their caries reduction or absence (especially IgA) in saliva. However, the findings are inconsistent, since it is difficult to link the general secretory antibody multifaceted system only with local manifestations in the oral cavity.

Soft dental plaque are quite pronounced enzymatic activity. Total number of enzymes found in dental plaque and dental plaque, more than 50, most of them are of microbial origin and are proteolytic enzymes. Besides them there phosphatase, neyraminaza, lactate dehydrogenase and other glycolytic enzymes of glucose degradation pathways. Under the action of microorganisms and enzymes of glucose and sucrose undergo enzymatic transformation. First, sucrose is converted to polysaccharides such as dextran and Levante who are reserved, and then hydrolyzed to glucose and fructose. Later they also phosphorylated by glycolytic converted to pyruvate. Further pyruvate can be reduced to lactate (lactic acid) by the enzyme lactate dehydrogenase, dekarboksylyuvaty in acetate (acetic acid) under the action of pyruvate decarboxylase or included in ttsykl Citric acid and form a number of organic acids, including succinic. Last under the influence of specific enzymes is converted to propionate. The combination of these acids (lactic, acetic, propionic) and forms a common set of dental plaque acid products that damage the mineral component of enamel. Among the predominantly lactic acid, which is produced mainly streptococci. Perhaps kariesogennoy properties of dental plaque is associated with it, because lactic acid has the ability to dissolve calcium apatite teeth even at pH above the critical value.

Role of microorganisms in caries development

The significant role of microorganisms in caries was confirmed by another work W. Miller, which enabled him in 1884 to formulate a chemical-parasitic theory of caries. Numerous subsequent studies has been very carefully studied microflora cavity and mouth, which allowed her to make a clear role in causing tooth decay.

Microbiological studies since the mid-XIX century., The cavity was discovered quite varied and numerous microflora. On the basis of the properties of its individual species, their prevalence in people with cavities and without their presence was revealed some relationship between different groups of microorganisms and caries. First it was the bacteria that can exist in an acidic environment and ferment carbohydrates, turning them into organic acids (called acidophilus and atsydohenni bacteria). Often these were some strains of streptococci, to a lesser extent – representatives of other species – lactobacilli, actinomycetes. The ability to ferment carbohydrates to form organic acids (lactic, acetic, propionic) is the most characteristic feature kariesogennoy microorganisms.

Streptococci of the cavity were first isolated in 1900 p., Was later found their quantitative predominance (50%) of all isolates kariesogennoy microorganisms. Assumptions about the important role of streptococci in the development of dental caries based on their quantitative predominance of the cavity, enzymatic characteristics, to identify them more than 50% of dental plaque and at different stages of caries lesions. According G.H. Bowden (1985), streptococci constitute 70% of the colonies, and veylonely neyseriyi – 15%, other microflora (dyfteroyidy, lactobacilli and other.) — 15 %.

Experimental conditions were simulated oral in vitro in the chemostat (so-called artificial mouth), which studied the peculiarities of growth and kariesogennoy of some microorganisms. The ability to cause demineralization of teeth has been established only in some species atsydohennyh microorganisms, for example, the strongest it has been in Str. mutans. Convincing experiments to prove the role of microorganisms in the development of caries were conducted F. Orland and colleagues (1964) Animal-hnotobiontah. It was found that in these sterile animals, even though they were on kariesogennoy diet (diet with a predominance of carbohydrate), caries not occurred. At the same time, the control group of animals that were ion-sterile conditions on the same diet, almost 100% of cases occurred caries. To clarify kariesogennoy activity of certain types of microorganisms, one strain was administered to food animals hnotobiontam (called monoinfikuvannya when the animal is only one type of bacteria). As it turned out, most types of microorganisms isolated from the oral cavity, causing no tooth decay, except kyslototvornyh bacteria (streptococci, lactobacilli and some others). Kariesogennoy highest activity in these animals was found monoinfikovanyh when entering streptococci, especially strain Str. mutans. This strain causes the most rapid development of caries with many (75%) of the affected teeth. Nowadays, the properties of this strain of streptococcus studied in great detail and shown its crucial role in the occurrence of dental caries.

Str. mutans often detected on the surface of the enamel, where it is a large proportion of the microflora of dental plaque. It prevails among organisms and materials from pits, fissures enamel interdental spaces, places that most often localization of caries. Very important is the fact that Str. Mutans normally absent from undamaged enamel surface if there is no plaque. There have been many studies that have established the prevalence of caries indices correlation with the presence of Str. mutans in the oral cavity. It was also demonstrated the ability of Str. mutans to adhere to the tooth surface by their synthesis of extracellular polymers such as dextran. It should be noted that other types of microorganisms can synthesize kariesogennoy of carbohydrates (especially from sucrose) vysokopolimerni glucans (dextrans, Levante), hyaluronic acid. These polymers enable the bacteria to adhere to kariesogennoy hard tissues of teeth (the enamel) and form the matrix of dental plaque.

In the experiment on animals P. Keye (1962) found that streptococci producing dextran, have a higher kariesogennoy activity. Such specific to Str. mutans properties, but less pronounced, with other strains of streptococci kariesogennoy (eg, Str. sanguis, Str. salivarius, Str. mulleri and some others). In the cavity and saliva of patients with dental caries bull found a significant number of lactobacilli. In the experiment had proved their high atsydohennist, atsydofilnist and ability to cause damage to the enamel kariyesopodibni. People with active caries process flow revealed a higher content of lactobacilli in saliva than healthy. Based on the determination of the number of bacilli in kyslototvornyh saliva test has been developed so-called laktobatsylyarnoho number, which can be an indicator of the activity of the caries process, or predisposition to the development of caries. Although lactobacilli are so active properties caot be considered the main microbial agent of dental caries.

Animal experiments, hnotobiontah been proven that kariesogennoy activity have only a few strains of lactobacilli (Lactobacillus acidophilus, Lactobacillus casei). It also established a major role in the destruction of lactobacilli dentine caries process. As you know, streptococci constitute almost half of all dental plaque microflora, lactobacilli – only 1%. Isolated from cavity streptococci have the ability to reproduce the experiment quickly, within a day after cultivation, they cause a sharp increase during plaque acidity – pH to 3.4. Lactobacilli reach a pH just 3 – 4 days, but so few of them able to give a very significant effect kariesogennoy. Assumptions about the possible role of other microorganisms in carious lesions of mainly based on the fact that many members of the oral cavity with active acidophilus and theoretically can cause demineralization of enamel. This assumption is evidenced by the fact polimikrobnosti dental plaque. Some of these species are resident microflora can cause caries in animals hnotobiontiv, but their role in the development of dental caries in humans has not been sufficiently clarified.

 

 In pediatric dentistry posluhovuyutsya common classification of caries lesions characterizing ¬ ing teeth nature of the flow, depth and localization.

I. Localization: fisurnyy (caries fissuralis); aproksymalnyy

(caries aproxsimalis); cervical (caries cervicalis); combined

location (buccal, labial, tongue surfaces).

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ocalization of cavities:

A – fisurnyy, B – tooth contact surfaces (aproksymalnyy);

B – cervical, G – circular

 

II. For deep lesions: initial (caries incipiens); over ¬ Neva (caries superficialis); median (caries media); deep (caries profunda).

 

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III. The nature of the clinical course of acute (caries acuta);

chronic (caries chronica).

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chematic representation of the cavity when

acute (A) and chronic (B) of the disease

 

IV. For consistency of occurrence: primary (caries primaria); secondary or recurrent (caries secundaria, seu recidiva). Along with the characteristics of carious lesions in a sepa ¬ mo tooth taken suggested (T.F.Vynohradova, 1978) to assess the activity of the caries process in the body as a whole. The author identifies three degrees of disease activity:

 And – compensated cavities;

II – subcompensated,

 III – decompensated (табл. 21).

The distribution of children into groups according to the degree of activity of the caries process is essential in organizing dental care pediatric population, particularly during sanation and preventive measures.

 

 

Table 21. Assessment of intensity of the caries process (with T. Vinogradova, 1978)

 

hat the depth of caries in children is the notional value, which depends not only on the size of the cavity, but also on the volume’s fixed camera. What is a “junior” tooth, the more volume it’s fixed camera and the in ¬ pared deeper cavities are.

Figure 28. Cross-section of the center of the initial caries (saL. M. Silverstone, 1981).

and – the surface layer, b-body lesions, in – dark zone; / – area hipermi-neralizovanoyi enamel.

And – look at polarizing microscope, B – schematic representation pas tomorfolohichnyh zones in enamel.

Pathological morphology of dental caries . Under a bright spot or initial caries with polarization microscopy detected cell lesions of enamel in the form of a triangle , whose base povernenado outer enamel surface ( SM Studevant, 1995, fig. 28). There are four zones: 1st – surface area, 2nd – body injury, 3rd – dark zone , 4th – transparent area.

The transparent zone is deepest . This area hipermineralizo – Vano enamel. It pores and voids located along the enamel prisms . Pore ​​volume in the clear zone of 1% , which is 10 times higher than in intact enamel.

The dark area is called so because they do not transmit polarized light will not ¬ . It has a large number of tiny mikroprostoriv under ¬ povnenyh air or gaseous substances that cause absorption with polarized light. Pore ​​volume in a dark area amounted ¬ vyt 2 to 4%. There is loss of the crystal structure of enamel. The width of the dark zone depends on the intensity of reminera – ation , while it may increase.

Body defeat – the largest volume area with obvious signs of de- mineralization. Pore ​​volume of from 5 % to 25% of the periphery to the center area. It can mistytytys bacteria, so that the size mikropros ¬ tors in the enamel enough to penetrate deep into them . Dissolution of enamel occurs along the lines Rechtsiusa as spaces along them relatively little mineralized .

Surface area hardly affected by caries . Pore ​​volume in ¬ it is not great compared with body damage ( meiishe U / O ) and mikrorenthe – tech is not different from the intact enamel.

The surface of intact enamel hipermineralizovana because of pos ¬ of continuous contact with the saliva , and the high content of fluoride ion in over ¬ Nebo layer of enamel making it more resistant to caries damaged ¬ tion versus located deeper layers. However prohresuvan ¬ ing caries process leads to the formation of the surface layer of enamel conical defects through which bacteria can enter inside the enamel.

Numerous electron microscopic study of enamel at different stages of carious spots revealed disorientation crystal ¬ sustainable hydroxyapatite , change their shape and size , the formation of atypical ¬ ing for normal enamel crystals.

At initial caries observed changes in the dental pulp : disruption of odontoblasts layer , change their shoots and homohe ¬ tion of the cytoplasm and fatty degeneration of connective tissue cells and extracellular material changes pulp.

At the surface and the average caries detected destructive ¬ tive and reactive changes of enamel and dentin.

Enamel- dentine combination of the least resistant to carious lesions, tooth decay because this place is spreading rapidly and carious lesion in dentin has a V- like shape with the apex directed toward ¬ tion pulp ( SM Studevant, 1995, fig. 29). This may explain the rapid spread ¬ You caries in teeth in children by plane (called the plane caries of deciduous teeth).

Pulp – dentinal complex responds to carious lesions intensification of mineralization of dentin to block dentinal tubules. This reaction is a result of enhancement of functional acciency odontoblasts in response to the demineralization process . Rose  riznyayut 3 types of responses to dentine caries process : 1) a reaction to a long, slow process of progressive low- acid demineralization lotnoyi 2) response to moderate- intensive process ;

3) reaction to the active, rapidly progressive carious process with high levels of acid demineralization

 

The determining factor in the implementation of protective changes in dentin with pulp zhythyezdatna with sufficient blood circulation and microcirculation .

In case of slow progress of caries pulp can restore demineralized dentin by dentin remineralization intertubulyarnoho . Dentin , which contains more mineral substances ¬ wines compared with intact called sklerozovanym . It is an obstacle to deepening the caries process , because it dentinal tubules almost completely blocked.

The second type of response is determined by a moderate dentin caries progression process. In the dentin contains a large number of pathogenic factors such as microorganisms,  ucts of their life, hydrolytic enzymes , remnants ¬ cial bacterial cells that can lead to degeneration and death of odontoblasts and their processes in the affected area and cause irritation of the pulp. Battered dentinal tubules forming the so-called dead path in dentin . In the pulp under the influence of substances coming through these ways of dentine formed secondary or so-called substitute Odon – toblasty (from undifferentiated mesenchymal cells). These cells produce reparative  HN (reactive ) dentin from carious pulp according to cell ( Fig. ZO ). This differs from secondary dentin dentin producing odontoblasts his Pul ¬ pi for life. Often the structure of reparative den portion irehulyarna and amorphous . Dentinal tubules there are no particular orientation .

The third type of response dentin detected in acute , progressive velocity com ¬ caries with high levels of acid . If the strength of destructive factors greatly exceed the protective capabilities of dentin and pulp, in which case the inevitable happens infection and inflammation of the pulp.

In carious tooth dentin distinguish 5 zones patomorfolohich -tion changes more clearly reveal the slow progress  bathroom decay, ie with its chronic course.

The first zone is the deepest – a zone of normal dentynu.U it is dentinal tubules with spikes odontoblasts , no crystals in tubules and bacteria.

 It is characterized by a large number of microorganisms in the dentinal tubules and destruction of organic structures dentin.              

The second zone – translucent dentine . This area demsheralizatsp intertubulyarnoho dentin and early formation of very thin crystalline ¬ hoists in the lumen of the tubules Significant damage processes odonto – blasts , but no bacteria in the tubules .

The third zone – transparent dentin. This layer of dentin characterszuyetsya further loss of minerals intertubulyarnym dentin and deposition of large crystals in the lumen of the tubules. It is softer compared to intact dentin . The bacteria do not. ¬ collagen fibers remain in the new structure as possible samovidnov -tion of the dentin in the living pulp.

The fourth zone – a zone of dentin with impaired histological structure. Characterizes hsya expansion and distortion dentin tubules ¬ them that are filled with a large number of microorganisms. There are very few minerals and collagen fibers destroyed.

The fifth zone – infected childr forth. This area was destroyed dent so steeped in lots of bacteria. The structure of dentin is not defined because there are no minerals and collagen fibers .

With an average caries are changing in the pulp. Odontoblasts layer thinned, vakuolizovanyy . Under the electron microen masse in odontoblasts revealed increased mitohondrp and weak korozvynena endoplasmic reticulum .

With deep caries , especially if it is an acute course, vidsuhni transparent zone ( sklerozovanoho ) zone intact dentin and dentin. Dentin bottom cavity substantially demineralized with varying degrees of degenerative changes in organic matter The reducnd in the pulp revealed significant changes bahahma authors lo ¬ tozhnyuyutsya initial phases of inflammation ( redness pulp).

 

Symptoms, diagnosis and differential diagnosis of caries of deciduous teeth

Clinic. Children of all ages caries has some features of the course. Anatomical and histological features of the structure of deciduous teeth, morphology and functional activity of the pulp at different stages of their development, and the general condition of the child’s body reactivity of an impact on the progress of caries. The clinical ob ¬ conservation suggests that the progress of caries in temporary teeth closely associated with the stage of development of the tooth.

 

Caries of deciduous teeth at the stage of root

A prerequisite for the development of early caries of deciduous teeth in children 1-3 years is disturbance of structure formation of hard tissues of deciduous teeth. It could be due to chronic somatic diseases are the mother before pregnancy, severe metabolic disorders in the mother during pregnancy (talk  sycosis 1st and 2nd half). Early lesions of teeth and their rapid Ruinuvannya often seen in premature infants, as well as those who are ill in the first months of life, and other infectious diseases (rickets, indigestion).

Feature of clinical caries of deciduous teeth in step form ¬ ing root is extremely sharp and acute course. Carious cheer ¬ tion localized mainly in the cervical area of ​​the upper incisors and the furrows of the first and second temporary molars. Tooth decay progresses rapidly , spreading on a plane and cover are resistant to decay in ¬ upper teeth ( incisors in vestibular , bumps in molars ). Vidznachayet ¬ be as rapid destruction of temporary tooth dentin because of its weak mineralization and lack of defensive reactions from mor ¬ folohichno and functionally immature pulp.

A characteristic feature of early caries lesions is the multiplicity of deciduous teeth and symmetrical arrangement of carious defects.

Despite the progress of active caries process , it is usually not accompanied by subjective feelings . This greatly complicates the differential diagnosis of various stages of decay between themselves and its complications , which most often manifested in the case of multiple disease.

Caries of deciduous teeth at the stage of root charac ¬ ryzuyetsya rapid transition in uncomplicated complicated. This is due to anatomical features of the structure of dentin and pulp in temporary teeth during this period : wide dentinal channel, a thin layer of mineralized dentin enough of pulp , a significant volume of the cavity of the tooth, the pulp horns that are close to the enamel- dentinal connections. Morphologically and functionally immature Pul ¬ pa at the stage of temporary tooth caot form sklerozovanyy (clear) and substitution ( reparative ) dentin that hinder the progression of the caries process .

In front of the upper jaw teeth are temporary and sometimes in molars caries can begin in the cervical area. Later, he distributed circular , covering the whole tooth. This form ca ¬ riyesu was called circular . Starting at anywhere near the neck of the tooth , often in the form kreydopodibnoyi spots fast decay com ¬ spans the neck, and then all temporary tooth crown . Ur ¬ tion of hard tissue is quite shallow , but quickly spread across the plane, all enamel layer existing disease that is easily broken. For ¬ Primary localization of the process observed mainly on the labial surface, in some cases – in language . The process can restrict ¬ tysya one temporary tooth surface , but quite often it spread ¬ ryuyetsya aproksymalni on the surface , leading to co- cast Ronco tooth. In the jaw , leaving only the roots of deciduous teeth , pulp flow in such cavities are usually nekrotyzuyetsya . ¬ Not rarely this age parents bring to the dentist when periodontitis has emerged in the area of ​​the front teeth of the upper jaw with porytsyamy on the gums, oozing pus or granulation of them ( see Figure 6 – see . Colored inset) .

Dental temporary molars in young children (2-3 years ) is characterized by acute course , is localized in the furrow and is intended to encompass beyond the enamel- dentinal connections , ie the depth of affection ¬ tion prevails medium and deep cavities. Carious cavity ¬ Nina light edge enamel thinned easily break off , dentin karioz ¬ tion cavity light, moisture, removable layers, and the whole process does not tend to limit.

 

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Caries of deciduous teeth formed at the stage of root

Acute initial caries – a transient stage of caries of deciduous teeth , so the hospital is rarely diagnosed . Carious spots are located in areas of typical localization of caries, ie fisu – Rahim , the aproksymalpyh surfaces of incisors and molars and in the cervical area. However, clinically carious spots often appear on the vestibular surface of the incisors temporary because these areas itso well foreseeable . Carious spot , usually covered with a thick layer of plaque, no subjective data , objective – while removing plaque and drying the surface area visible white enamel ¬ of color, which have lost their natural luster. Acute primary tooth can become chronic course or go into sharp ¬ ing surface caries.

Superficial caries is more acute course and with the consequence of ¬ strictly initial caries. Burying caries in enamel ¬ nated accompanied by the appearance of carious defect, but that does not cross the enamel- dentinal connections. Localization of acute superficial ¬ carious cavities corresponding to the localization of spots. Defects of enamel ext re ¬ visible on clinical examination and have the appearance of areas Cray – dopodibno modified enamel with visible destruction of its structure. When probing determined rough , softened surface. Complaints are usually absent. Older children may complain of the action of chemical stimuli ( sour, sweet).

Chronic superficial caries of deciduous teeth on the stage of formation of roots is rare. There is a lack of subjective sensations. It appears during the clinical examination of the child. Carious defect looks like a dark brown plya ¬ we defective enamel sensing does not cause painful sensations.

Average caries . Acute secondary caries of deciduous teeth – one of the most common clinical forms cavities formed at the stage of root. The child may complain of delay of food between the teeth sensitivity ¬ lyvist in the event of chemical and thermal stimuli. Sometimes complaints can ¬ may be available. During the physical examination detected carious cavity with a narrow inlet . Pidryti edge enamel with matte white. Dentin that fills a carious cavity ¬ nynu , light yellow or yellow color , soft, removable layers for an excavator . In aproksymalniy surface caries may cover the entire surface with little depth. In raids, formation of thin edges of enamel carious cavity may have broad ¬ cue inlet . Probing bottom and sides cavity is usually painless. It should be remembered that these sensing in such cases you ¬ is not always objective , due to the psycho-emotional state of the child during treatment.

Chronic secondary caries is localized mainly in aprok – being maximum surface , at least – for chewing and cervical . The clinical course of this form of caries in temporary teeth bezsymp -volume . Complaints may be the presence of the cavity or delay food between the teeth. Cavity has a wide inlet , its walls and floor are covered with dense pigmented dentin . Areas ¬ ment walls and floor painless.

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Sometimes somatic healthy children revealed significant planar defects of hard tissue (called planar cavities ) with dense, shiny, smooth in probing the dentin . They are located on the chewing surfaces of temporary molars. This condition can be regarded as suspended caries ( tooth decay stationary ). It could be due to improved overall physical health di ¬ fences and changes kariesogennoy situation in the mouth.

Deep cavities formed at the stage of temporary tooth root is more acute course. Subjectively children may complain of pain as a result of mechanical or thermal stimuli.

During the physical examination should carefully examine the causal tooth, pay attention to the intensity decay and deter ¬ cheats history of the state of general physical health of the child. Cavity hfy acute deep caries localized within prypulphyuvoho dentin. Yii depth in temporary teeth are smaller than regular , because of the anatomical and topographical features of the structure of deciduous teeth.

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It should take into account the location of the cavity. When placing the cavity in aproksymalniy surface where the layer of solid fabric is quite thin and the distance to the pulp insignificant complications of caries development is faster. Because acute course of decay with a cavity on the surface aproksymalniy temporary tooth is often a sign of complicated caries .

If the active current caries in temporary teeth do not have time to be produced vicarious ( reparative ) dentin from the pulp, not pronounced protective hardening in the dentin . Dentinal channel are broad processes of odontoblasts quickly destroyed tubules filled with a mixed bacterial flora , as irreversible changes in the pulp of deciduous teeth can be observed at clinically shallow cavities. Therefore, the diagnosis of acute deep caries of deciduous teeth should be set very carefully after a thorough diagnostic differentiation tion of its complications.

Chronic deep caries in temporary teeth in children under the prevailing root does not occur often. It can be diagnosed in somatic healthy children with low intensity of the caries process . Chronic tooth decay is characterized by free ¬ progression , the formation of dense , sklerozovanoho dentin due to activation of protective function of morphologically mature pulp. Subjective complaints during the course of this decay are absent. Objectively manifested in the tooth carious cavity with a wide inlet . Carious dentin thick, is dark brown or black color -ing , excavators removed hard nry sensing probe is not retained and easily glides over the surface .

In addition to acute and chronic course of caries in temporary teeth in the clinic are so-called intermediate forms between the classical signs of acute and chronic caries. Thus the observed  yutsya moderately severe pigmentation and decalcification of dentin cavities. Dentin is the yellowish – brown color , but the texture is not hard, as in the case of chronicity.

Caries in deciduous teeth root resorption stage

At the stage of root resorption of deciduous teeth caries zustrichayet not be often. The clinic is mainly diagnosed complication of tooth decay in the form of chronic pulpitis and periodontitis. However, the soma- cally healthy children carious process in temporary teeth may have a typical chronic course. For deep affection prevails ce ¬ vious and deep cavities , rarely observed surface caries as pigmented spots, which is localized on the surface of polar vestybu temporary canines. Localization dominated aprok – being maximum caries. According to TF Vinogradova (1988 ), localization of caries on surfaces aproksymalpyh temporary molars and upper incisors observed in almost 60 % of cases, whereas in the cervical area , it is found only in 5 % of patients.

Acute course of caries of deciduous teeth on the stage of resorption comrenya rarely diagnosed and usually in children with common catfish  cal diseases and reduced immune reactivity. In this appeal no hope stimuli .

Thus , the flow of caries of deciduous teeth has certain laws ¬ nomirnosti corresponding stages of their development.

Differential diagnosis of caries of deciduous teeth. If the diagnosis of caries of deciduous teeth occurs, usually without much difficulty , the differential diagnosis of non- complicated and complicated caries of deciduous teeth – extraor ¬ but responsible and difficult task that confronts every stoma ¬ tolohom pediatrician . Age of child difficulties contacting, ¬ insufficient classification of their feelings baby occasionally make this task extremely difficult. Therefore, differential diagnosis of caries of deciduous teeth is based mainly on data from the objective circumstances -tion of the child. At the same time pay attention to the depth and location of the cavity , the color and texture of the affected dentin, espe ¬ cially condition dentin layer at the bottom of the cavity , the condition of the mucous membrane of the gums and transitional fold area carious tooth.

For differential diagnosis of caries of deciduous teeth develop m’yakshenyy dentin should completely remove an excavator or Kulas by boron. If this open cavity of the tooth within the softened dentin , the diagnosis is not difficult to figure out . The presence of Pulpi bleeding and reacts to sound , confirms asymptomatic tion of chronic fibrous pulpitis. If the probe conattraction cavity with pulp chamber painless, it may indicate a chronic gangrenous pulpitis or chronic granulating periodontitis. To confirm this , carefully inspect and palpate soft gum tissue and transitional folds in the area of ​​the affected tooth. The presence of even slight hyperemia rederivatives folds or fistula on the gums evidence of asymptomatic periodontitis temporary tooth. The final diagnosis is established  regimenting after X-ray examination.

Symptoms, diagnosis, differential diagnosis of caries of permanent teeth

Clinic. Caries in permanent teeth of children as the mass of a certain person ¬ portunities course. They are mainly associated with the stage of formation of permanent teeth and mostly – the state of health of the child that EID ¬ Nacha its immunological reactivity. The frequency lesion 1st place went to the first and second permanent molars of the lower jaw, on the 2nd place – these very teeth of the upper jaw. They were subjected to shock upper incisors and premolars . Less commonly observed lesions of the upper canines and lower premolars. Quite resistant to caries lesions lower incisors and canines .

In children, severe forms dominate the flow of permanent teeth caries is caused by the incomplete mineralization of hard tissues nin permanent teeth that have just erupted , and the hysnoyu sufficient for the function of the pulp during the completion of their formation. The less time since the eruption of the tooth to its defeat , the sharper and more rapid progress of caries. During the generated correlationtion of permanent teeth more frequently than acute , there is an intermediate and chronic caries. This is due to the stabilization of the structureri enamel and dentin and pulp of functional maturity that the ability to deter rapid spread of the caries process by making you  substitution and transparent dentin. Acute progress of caries in adolescents , covering a large number of permanent teeth, indicating the presence of immune deficiency in the body. Therefore, it is obligatory  careful examination of these children are not pediatricians ( Gastroenterologist, endokry -gies et al. , Fig. 7 – dyv.kolorovu inset).

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Localization of carious lesions is also associated with stage forming tooth. In teeth with immature root caries locale  zuyetsya mainly iatural cavities and furrows crownsing . Furrows – enamel cover this area , formed by the merger of molars or premolars hills that form  Xia and mineralizuyutsya separately. Some pieces of landing tooth crowns that are saturated with mineral salts, increasing merge and form a single occlusal surface of the tooth . In places of connection and form furrows , folds and holes. Thicker enamel cover at the junction of some relatively small hills and bottom grooves separated from the dentin to priorities would need a thin layer of enamel ( 0.5-0.6 mm). At the time of tooth eruption in a child furrows are less mineralized areas we crown. Retention of food in the furrows, which are directed at nesfor – tooth deep and wide, resulting in early cheer  tion of caries (Fig. 31). During the formation of roots  localization of caries in the furrows observed much less frequently , giving aproksymalnomu location in carious cavities in permanent teeth of all groups.

 

Chart. 31. Caries development in the area of ​​fissures (polarization microscopy).

A – tooth decay begins in the region of the inlet in the fissures and localized

only within the enamel of the arrow.

B – due to lesions of enamel fissure caries at the bottom of the process extends to the dentin

As for the depth of the lesion , the permanent teeth in children can identify all forms of dental caries : initial , superficial , medium and deep ¬ Bokij . However, the clinic is dominated by medium and deep cavities.

Initial caries or carious enamel demineralization (caries incipiens, seu macula cariosa), clinically characterized by a change of co ¬ loru in a limited area of enamel – the appearance of spots. Children of glass ma ¬ often has a white matte , less brown or black ¬ ing . In the initial stage can often diagnose caries on the exposed surfaces of teeth – vestibular and cervical . So if you start talking about tooth decay, or cavities in stage spot, ¬ tion should mean caries vestibular surfaces of front teeth or cervical surface of all the other teeth. It should be borne in mind that each cavity regardless of its location must re ¬ blowing an early stage in the form of patches.

Spot white larger than 1 mm would take ( H.N.Pahomov , 1974) for the first clinical stage of decay. Increasing the size of spots is accompanied not only by an increase in the area of enamel lesions , but also the degree of destructive patomorfolohich – change.

When gaps larger than 3 mm of the pathological process ¬ fully covered layer of enamel , causing changes in the enamel- den combination hospitable and even in dentin . Changes of organic matter in ¬ shkodzhenoyi zone material .

Brown spots are fundamentally different from whites ¬ we greatly destructive changes in organic matter of enamel. When you ¬ roof of hard tissue lesions always drawn into the process of enamel- dentine connection and turns reaction ( sclerosis) of dentin bordering the center of the pathological process. The depth of the lesion responsible Square ¬ charge spots.

Acute initial caries in children are usually not complaints. His doctor finds during the examination of the teeth. Plot naychas ¬ lesions more often covered with plaque removal after which exhibit piece white enamel that have lost their natural luster. Returning ¬ hnya enamel is smooth , sometimes a little rough , but painless and very hard. In permanent teeth kreydopodibni spots detected in the cervical area incisors and first permanent molars , and in children 12-15 years – in the area of the necks of the canines, premolars , molars less .

Acute initial caries is diagnosed more often in children with III degree of activity of the process , which includes a large number of teeth, INCO ¬ ly even all teeth. This may indicate a significant shift in a state of systemic and local immunity of the oral cavity, which ob ¬ rihayutsya chronic diseases of internal organs and body systems.

Diagnosis of acute initial caries carried out first of all ¬ ed visually. To do this, clean the enamel surface of teeth on the fly ¬ thoroughly dried air stream. The affected area becomes opaque enamel shade resembling enamel after mordants state ¬ ment when working with composites .

For the differential diagnosis of acute initial caries lesions from non-carious hard tissues ( enamel hypoplasia , flyuo develop ¬ ) is most often used method zazhyttyevoho (living ) in ¬ staining with 2 % aqueous solution of methylene blue ¬ nd . For this enamel surface previously cleaned, treated with hydrogen peroxide and dried. Teeth are isolated from saliva and put them in the dye solution for 2-3 minutes. Then dye wash water stream. Damaged areas of enamel in acute initial caries ¬ tural , unlike hypoplasia and fluorosis , stained with varying intensity. Assess the color on a 10- point scale. As the dye can be used as 0.1 % aqueous solution of Me ¬ tylenovoho red.

The method of electrometric diagnosis of caries and com ¬ plex devices to detect initial caries not only on the types ¬ IIR sections of the tooth , but also in the furrows ( V.K.Leontyev , 1983). Method of soil ¬ tuyetsya the ability carious tissues conduct electrical current of varying size depending on the degree of injury.

In addition, in order to diagnose the initial decay can be per- stosovuvaty method stomatoskopy ‘ ultraviolet irradiation, based on the effect of luminescence hard tissues : healthy tissue lyuminestsiyuyut light green. Dental promotes ¬ Sonny luminescence of said hard tissue . It is larger, the deeper the pathological changes in the structure of the enamel.

Recently stomatoskopichnoyi for diagnosis of caries using fotopolimeryzatory that they are used to working with composite photopolymer materials .

Differential diagnosis of acute initial caries of enamel hypo ¬ plaziyeyu (especially thistle shape) and fluorosis are in tabl.22 .

It is extremely difficult to differentiate acute initial caries fisurnyy permanent teeth , and insufficient mineralization fissures of premolars and molars. The basic clinical research method – probe ¬ tion – does not show significant differences , as demineralized enamel and poorly mineralized equally tempered and rough during probing.

During the formation of roots of permanent teeth grooves naychas ¬ more often pigmented , so a correct assessment of mineralization and diag ¬ teak initial caries – important task for dental ha pediatrician . It should be borne in mind that most of them mineralizova fissures , even in the presence of the pigment should not be regarded as an initial or superficial caries, as mineralized as connector may be resistant to decay during the whole functioning of the toothtion . Pigmented groove in which the probe is not delayed nor deepens, should not be considered as initial caries. In furrows  priorities would need to see a stabilization of caries often requires no plom  accommodated.

Table 22. Differential diagnostic signs of acute initial caries, enamel hypoplasia and fluorosis

Signs

caries

Hypoplasia

fluorosis

Time of

After the eruption

Before the eruption

Before the eruption

Affected teeth

Equally and permanent and temporary

Preferably constant (very rarely temporary)

Constant

arrangement

Furrows and other depressions con-stroke and Coming-term tooth surface

Vestibular and lingual surfaces

Vestibular and lingual surfaces

Number of spots

Single, rarely more

Most single

Multiple

Permeability to dye

significantly increased

Do not change

Do not change

Changes spots

Rarely disappears, time-more often

  On-site there is a carious cavity

persists

Sometimes it may be reduced, but more remains for life

Dependence of fluoride in

water

I will smite the growth-ness at low concentrations of fluoride in drinking water

Do not depend

The emergence of local self-tsevostyah where sub-vyschenyy content of fluoride in drinking water

 

Superficial caries in permanent teeth of children occur in place of white or pigmented spots due to progression of destructive changes in the enamel. Characterized by the softening of the affected enamel is removed with little effort excavator. Most children at this stage of the pathological process any complaints are observed. Some of them complain of intermittent pain from chemical irritation ers – sweet, salty, sour.

If in the case of excavation stripped enamel dentine is destroyed enamel- dentine combination , then the tooth should be considered for this vious .

Clinically surface caries is under examination in upper tooth ( enamel discoloration ) and during probe : sharply expressed weighted roughness, probe delay , defects of enamel.

Differential diagnosis of acute superficial caries should be conducted with acute primary and secondary caries, alveolar form of systemic or local enamel hypoplasia , as well as erosive form of dental fluorosis. Hot surface caries differ from the initial acute complete destruction of the surface layer and Society ¬ lyblennyam pathological process in the enamel. Unlike acute , intermediate decay is characterized by a sharp surface retentiontion intact enamel- dentinal combination that at the average caries always destroyed. With an average caries cavity commonly lyblyuyetsya to dentin, enamel- dentinal sensing communication in case of acute flow causes pain.

Cellular form of enamel hypoplasia differs from the initial decay multiplicity of defects and symmetry of their location. No signs of hyperesthesia observed. Hypoplastic defect charac teryzuyetsya correct spherical contours, edges are smooth out  Eugene. The bottom of the defect is always smooth and shiny .

Local hypoplastic enamel (tooth Turner ) , as opposed to surface caries, enamel defect characterized by irregular shap we often pigmented , located on the mound pre- molars or on the cutting edge of front teeth . The bottom of the de defects formed by a thin layer of enamel or dentin sklerozovanoho . This non-carious lesions of teeth relatively early caries complicated .

Erosive form of endemic fluorosis as surface caries is characterized by a defect within the enamel. However, when defluorosis enamel defects can be located on any surface of the tooth, including resistant to decay. Caries process in such defects is practically not observed. Since erosive form of fluorosis occurs when the use of drinking water with high fluoride content (in mg / l or more ), the signs of fluorosis manifest in the majority of children who live in the region.

Average caries of permanent teeth in children diagnosed naychastist . For this form of caries process violated the integrity of the enamel- dentinal connections, but on pulp chamber remains unchanged fairly thick layer of dentin. In most children , this forma permanent teeth caries complaint is not so diagnosed, usually in the rehabilitation of the mouth or teeth review of medrem dentist.

Cavity is characterized by a small input from vorom . Occasionally affected furrow comes only probe that there is attrymuyetsya . The edges of the enamel that covers the entrance to the cavity, can be kreydopodibno changed, especially in the in aproksymalnyh upper front teeth. Therefore, to determine the depth of the lesion den  portion , and thus tooth decay and form is possible only after preparuvaning cavity , which is a sensitive (if performed without anesthesia ) through stimulation enamel- dentinal  combinations of boron.

For older children who are healthy somatic , in the teeth of the external check vanym root caries diagnosed chronic middle . The character  it for it is a slow progression in dentin walls and bottom carioznoyi cavity rather dense, brown. As a rule ¬ lo , these children are diagnosed form of activity offset carioznoho process.

Differential diagnosis . Average caries of permanent teeth in children should be differentiated primarily with deep caries , as well as chronic peryudontytom . During the differentiation state trough medium and deep cavities should pay attention to the depth of the cavity after preparation. It should take into account  wool in permanent teeth with immature root pulp relatively larger volume because carious cavity at a relatively lower its depth can be located close to the pulp. Acute invincibly  cue caries tooth is more sensitive to thermal and mechanical stimuli. Cool in the tooth affected by acute deep caries  bokym , there is pain that passes quickly after removal podraznyka . Sounding pretty thin bottom cavity in case of acute deep caries sensitive because of the proximity Pulpi , while the average for acute caries more sensitive sensing is a wall cavity.

Chronic secondary caries in children sometimes have differentiated  yuvaty chronic form of periodontitis. The peculiarity of the current re- riodontytu children may be its development in a closed cavity of the tooth. This odiyiakovoyu measure applies to temporary and permanent teeth. When differentiating pay attention to the color of the tooth , and the reactiontion of the child during cavity preparation ( if performed without anesthesia ). In chronic periodontitis, especially permanent tooth , its color changes to gray. Prepaing enamel- dentinal connection is not accompanied by painfully  we sensations. These characteristics are the basis for radiographs of the affected tooth, which allows you to put the final diagnosis.

Radiography is used to diagnose and sometimes in hovanyh cavities located in aproksymalnyh in  upper teeth and invisible during the inspection .

Deep caries in permanent teeth of children diagnosed frequently. This stage is characterized by the decay that the pulp remains the halls thin layer of dentin. Pathologic changes detected in odontoblasts and pulp basic substance , congestion and round -vascular inflammatory infiltrates. Cavities located within prypulpovoho dentin. Therefore, probing her bottom quite sensitivelyve . Children may also complain of pain on thermal and mechanical stimuli are  , which passes quickly after their removal.

Differential diagnosis . Differentiate acute deep caries of permanent teeth should first of chronic fibrous pulpitis , pulp hyperemia and limited acute pulpitis , chronic form of periodontitis.

The main difference from caries pulpitis is the lack of wide  free twinge . Therefore, when collecting medical history should carefully examine whether there was a ever a pain. In addition, for Pulpitta characteristic pain of longer stimuli. Therefore, during the protermodiahnostyky maintenance should pay attention to the duration of paining sensation after removal of the stimulus. If the pain does not decrease immediately ¬ peared , and takes some time, it is a sign of pulpitis. After cavity preparation should carefully examine its bottom. The presence at the bottom of the cavity area pronounced softening of the dentin , and sharp pain in the sensing area that is featured ¬ ing chronic fibrous pulpitis. During the differential diagnosis invincibly ¬ whom caries and chronic fibrous pulpitis should be considered and each state physical health of the child. Certainly, children who HVO  riyut chronic diseases of internal organs, with sub- or – Dekom pensovanu form of caries activity , often justified by the diagnosis “chronic fibrous pulpitis ” with considerable depth of carious cavity nyny .

For the differential diagnosis of permanent teeth to the current root method can be applied elektrodontodi – agnostic. Indicators elektrozbudzhuvanosti pulp normally not ne ¬ exceeds 2.6 mA. In the case of pulpitis , this figure increased to ¬ schuyetsya 25-40 mA.

literature:

1. Diagnosis and Caries diagnosis dyfferentsyalnaya teeth & its complications. Textbook. / [VF Mihalchenko, LI Rukavishnikova, N.N.Tryholos] – M.: AOP Park Jangar “2006. – 104 p.

2. Kononenko JG Local anesthesia in outpatient, dental interventions / JG Kononenko, M. Rozhko, GP Ruzina / / Ivano-Frankivsk, 2006 – 295 p.

3. Preventive dentistry: Tutorial. / [MF Danilevsky, A. Borisenko, A. Politun, LF Sidelnikova]. – K.: Health, 2004. T. 2. – 400 pp.

 

 

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